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NANNING, Sept. 1 (Xinhua) -- In south China's Guangxi Zhuang Autonomous Region, an area renowned for its history of musical folklore, local residents have found a unique way to spread knowledge regarding AIDS prevention."All people unite to fight AIDS! AIDS will stay away if we are equipped with AIDS knowledge," state the lyrics of one song that was penned by local songwriters as part of a local anti-AIDS campaign."We asked eight local singers to write and record songs about the disease. We think this is an effective way for local people to acquire AIDS knowledge," said 55-year-old Huang Zhanghui, a resident of the village of Shantun. He said that the songs are played through loudspeakers in his village every evening.The songwriting campaign has been effective because folk songs are a traditional artform in the region, making them more readily understandable and acceptable for local residents, Huang said.Guangxi has been one of the hardest-hit areas in China in terms of the number of AIDS infections discovered there, ranking only after central China's Henan Province. More than 76 percent of the infections are sexually transmitted, according to Ge Xianmin, an official from the AIDS prevention office of the Guangxi regional government.Bama County, which administers Shantun, is known for the longevity of its residents. Nearly 2,500 of its residents are more than 80 years old, and another 81 have celebrated their 100th birthdays."We will not let AIDS threaten this tradition," Huang said.Huang was selected to be the head of the village's AIDS prevention office just two months ago. In addition to the creation of folk songs, the office spreads AIDS prevention knowledge through text messages and publicly screened films.

SUVA, Sept. 21 (Xinhua) -- The Secretariat of the Pacific Community (SPC) stressed on Wednesday that sustainable use and management of forests and tree resources will remain its important focus for the foreseeable future.Sairusi Bulai, a forestry team coordinator of SPC's Land Resources Division (LRD) told a regional workshop currently underway in Fiji's western tourist city of Nadi that "our main issue was and continues to be the lack of adequate resources to enable countries to effectively implement sustainable forest management.""Therefore, we are very fortunate that we have this opportunity to discuss findings and recommendations of the mission, which the UN-REDD Program has undertaken in the Pacific earlier this year," he added.REDD+ is a new international mechanism to compensate developing countries for reducing their rate of deforestation and forest degradation and increasing their carbon stocks.Developing countries participate on a voluntary basis. They provide technical and financial assistance to enable eligible countries to meet the requirements for future participation. Once a country begins participating, it receives compensation for its verified reduction in carbon emission from forest activities.The UN-REDD Program is a collaborative partnership of the United Nations Development Program (UNDP), the United Nations Environment Program (UNEP) and the Food and Agriculture Organization (FAO) to support countries in the preparation of this mechanism.Bulai encouraged participants to take advantage of the excellent opportunity to discuss REDD+ experiences in the region and to link up with the experts in attendance.REDD+ gives an excellent opportunity to create better awareness and understanding amongst all stakeholders, including resource owners, so that informed decisions are made to avoid exploitation, he said.Bulai, meanwhile, stressed on the critical importance of multi- sectoral as well as multi-stakeholder based approaches to forest management to effectively reduce deforestation and forest degradation.

SAN FRANCISCO, June 13 (Xinhua) -- Social networking giant Facebook will likely go public in the first quarter of 2012 with a valuation that could top 100 billion U.S. dollars, U.S. media reported on Monday.In a report, CNBC quoted people familiar with the matter as saying that Facebook could submit filing to register its securities with the U.S. Securities and Exchange Commission (SEC) as early as October or November this year.People who are on Wall Street and track this information told the business news television channel that they think the Facebook initial public offering (IPO), if and when it happens could value the company of more than 100 billion dollars.A factor in the company's IPO timing is the SEC's requirement that companies must disclose financial information if they have more than 500 private investors."The company has until the end of April 2012 to disclose their financials, but they may just want to get ahead of that by doing a formal initial public offering, I'm told. And that could happen in the first quarter of the year," said CNBC Wall Street reporter Kate Kelly.Facebook is also facing internal pressure as employees have not been permitted to sell their private shares on the secondary market since last spring. An IPO would make it easier for employees to monetize their shares, said Kelly, citing sources.Facebook shares have been traded in private markets such as Sharepost.com, which puts the social networking company's valuation at 85 billion dollars.Meanwhile, latest data show that Facebook is losing users last month in the United States, Canada and several European countries, indicating that the company could have hit the limits of expansion in its mature markets.

CANBERRA, Sept. 7 (Xinhua) -- A team of Australian and U.S. scientists on Wednesday said they discovered a genetic defect, which can lead to Leigh syndrome, a rare disorder which affects the central nervous syndrome.The scientists tested more than 1000 genes by encoding proteins active in two individuals who suffer from the illness. They used a new technique known as next-generation DNA sequencing to examine the genes.The gene they discovered encodes an enzyme which is found in the mitochondria which are subsets of cells. Without this enzyme the mitochondria do not translate proteins efficiently, and this then causes Leigh syndrome.According to David Thorburn, from the Murdoch Children's Research Institute in Melbourne, the discovery shows the enormous potential of the new technology."These findings demonstrate the ability of sequencing technologies to improve diagnosis," Professor Thorburn said in a statement released on Wednesday."Although it isn't clear in the case of Leigh syndrome whether the precise molecular diagnosis will necessarily lead to therapies, the current findings represent a meaningful service."He added that diagnosis of the disease along with its specific genetic cause can also be informative about the risk a couple has of having another affected child. The diagnostic information can help in decisions about whether and how to pursue alternative means of having children, for instance through the use of donor sperm or eggs.The research team consisted of scientists from Australia's Murdoch Institute as well as the Broad Institute in the U.S.In Leigh syndrome, infants are born apparently healthy only to develop movement and breathing disorders that worsen over time, often leading to death by the age of three. The problem is that the mitochondria responsible for powering their cells cannot keep up with the demand for energy in their developing brains.

WASHINGTON, Sept. 12 (Xinhua) -- An enzyme that appears to play a role in controlling the brain's response to nicotine and alcohol in mice might be a promising target for a drug that simultaneously would treat nicotine addiction and alcohol abuse in people, U.S. researchers find.Over the course of four weeks, mice genetically engineered to lack the gene for protein kinase C (PKC) epsilon consumed less of a nicotine-containing water solution than normal mice, and were less likely to return to a chamber in which they had been given nicotine. In contrast, normal mice steadily increased their consumption of nicotine solution while the mice lacking PKC epsilon did not.The study conducted by researchers at the Ernest Gallo Clinic and Research Center, affiliated with the University of California, San Francisco, appeared Monday in the online edition of the Proceedings of the National Academy of Sciences.In normal mice, as in humans, nicotine binds to a certain class of nicotinic receptors located on dopamine neurons, which causes dopamine to be released in the brain. Dopamine creates a feeling of enjoyment, and thus prompts a sense of reward. Researchers found that mice lacking PKC epsilon are deficient in these nicotinic receptors.The finding complements earlier research in which researchers found that mice genetically engineered to lack the PKC epsilon enzyme drank less alcohol than normal mice and were disinclined to return to a chamber in which they had been given alcohol."This could mean that these mice might not get the same sense of reward from nicotine or alcohol," said Gallo senior associate director and investigator Robert Messing. "The enzyme looks like it regulates the part of the reward system that involves these nicotinic receptors."The reward system is a complex of areas in the brain that affect craving for nicotine, alcohol and other addictive substances.The next step in the research, said Messing, would be to develop compounds that inhibit PKC epsilon. The ultimate goal, he added, would be medications that could be used "to take the edge off of addiction by helping people get over some of their reward craving."