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DAMASCUS, Sept. 21 (Xinhua) -- The national archeological expedition recently discovered a gigantic building built with large-size hewn stones that date back to the Roman era, Syrian Arab News Agency (SANA) said Wednesday.The building, the thickness of its walls up to 1.5 meters, was found at Qumet Nibal site in the northeastern province of Raqqa, said SANA.Ibrahim Kheir Bek, head of the archeological department in the area, told SANA that the excavation unearthed a 6-meter-long entrance to the building, adding that works are still underway in the site.A day earlier, the national archeological expedition discovered a mosaic dating back to the 6th century A.D. in northeast Syria, SANA said, adding that the rectangular mosaic was found at Hwaija Halawa site on the banks of al-Assad Lake in Raqqa province.Ayham al-Fakhri, head of the national expedition, was quoted by SANA as saying that the expedition uncovered 58 square meters of the mosaic with various colorful geometric shapes.

SAN FRANCISCO, Sept. 2 (Xinhua) -- A late-season potato blight has been detected on farms in Alaska, only the fourth time the disease appear in the U.S. state since 1995, local media reported on Friday.The disease was found this week in the fields of potato producers in cities of Delta Junction and Palmer, according to the Fairbanks Daily News-Miner, Alaska's second largest daily newspaper.Farmers in both fields are working to prevent the spread of the blight, a fungus-like disease that can kill plants in the field or cause potatoes to rot in storage, the newspaper reported.Growers have been asked to kill all affected plants within 100 feet, and plant samples from both sites will be examined to identify what type of late blight is involved.Experts said that blight can be controlled through the use of fungicides, but the cool, damp weather this summer in the areas has created an ideal environment for the disease to spread.Although Alaska's potato crop is tiny compared to other U.S. states, it has been viewed as a possible global source of seed potatoes in recent years because it is largely free of disease.A local university has explored partnerships with Chinese growers to export certified seed potatoes, noted the Fairbanks Daily News-Miner report.

THE HAGUE, Aug. 16 (Xinhua) -- ESBL-enzymes, which are known for their ability to stop the effects of antibiotics, can directly pass from chicken to people, said a research published on Tuesday.Veterinary researcher Dik Mevius of the Wageningen University released the finding in the magazine Resource.The research group led by Mevius has isolated six ESBL-genes varieties and compared them to bacteria found on hospital patients, on super market poultry and in laying batteries. In 35 percent of all cases, they discovered a match of 100 percent.Bacteria like Klebsiella on E. coli can produce the ESBL-enzymes. A healthy person will not feel any effect, but people with a lower immune system can get very sick. In areas like a hospital, the infection can easily spread."The main concern is that the enzyme resists the most used antibiotics, which makes the treatment of infected persons difficult," said Mevius.According to Mevius, 94 percent of supermarket poultry carries the enzyme, but all enzymes will be killed during the heating process of cooking.

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."

LOS ANGELES, June 24 (Xinhua) -- Near-Earth asteroid 2011 MD will whip past Earth on June 27, but will not pose any threat, NASA's Jet Propulsion Laboratory (JPL) said on Friday.The asteroid will pass only 12,000 kilometers (7,500 miles) above the Earth's surface at about 9:30 EDT, according to JPL in Pasadena, Los Angeles.This small asteroid, only 5-20 meters in diameter, is in a very Earth-like orbit about the Sun, but an orbital analysis indicates there is no chance it will actually strike Earth on Monday, JPL said.If a rocky asteroid the size of 2011 MD were to enter Earth's atmosphere, it would be expected to burn up high in the atmosphere and cause no damage to Earth's surface, said JPL.The accompanying diagram gives a view of the asteroid's trajectory from the general direction of the Sun. This view indicates that 2011 MD will reach its closest Earth approach point in extreme southern latitudes (in fact over the southern Atlantic Ocean), according to JPL. The incoming trajectory leg passes several thousand kilometers outside the geosynchronous ring of satellites and the outgoing leg passes well inside the ring, JPL said.For a brief time, it may be bright enough to be seen even with a modest-sized telescope.One would expect an object of this size to come this close to Earth about every six years on average.The asteroid was discovered by the LINEAR near-Earth object discovery team observing from Socorro, New Mexico.