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WASHINGTON, June 8 (Xinhua) -- Hundreds of small genetic variations are associated with autism spectrum disorders, including an area of DNA that may be a key to understanding why humans are social animals, according to a multi-site collaborative study led by researchers at Yale University.To be published Thursday in the journal Neuron, the study reinforces the theory that autism, a disorder that develops in early childhood involving impairments in social interaction, language deficits and distinctive behaviors, is not caused by one or two major genetic defects, but by many small variations, each associated with a small percentage of cases.The study looked at more than 1,000 families in which there was a single child with an autism spectrum disorder, an unaffected sibling and unaffected parents. The team compared individuals with autism to their siblings to determine what types of genetic changes distinguished the affected child from the unaffected child.One of the most intriguing of these findings points to the same small section of the genome that causes Williams syndrome -- a developmental disorder marked by high sociability and an unusual aptitude for music.In autism, there is an increase in the chromosomal material, an extra copy of this region, and in Williams syndrome, there's a loss of that same material," said lead author Matthew State. "What makes this observation particularly interesting is that Williams syndrome is known for a personality type that is highly empathetic, social, and sensitive to the emotional state of others. Individuals with autism often have difficulties in the opposite direction. This suggests that there is an important key in that region to understanding the nature of the social brain."State and his team also found about 30 other regions in the genome that are very likely contributing to autism and are focused on about six of those regions that showed the strongest evidence."We're now moving on to a second phase of the study looking at an additional 1,600 families and should be able to identify multiple new regions that are strongly implicated in autism," he said.

JIANGYIN, Jiangsu, July 1 (Xinhua) -- China's manned deep-diving submersible, the Jiaolong, embarked on a journey on Friday during which it will make a 5,000-meter dive in the Pacific Ocean.During the dive, the submersible will undergo several operational tests in which it will take photos, shoot video, survey seabeds and take samples from the ocean floor, according to Jin Jiancai, deputy director of the submersible's diving test program team.The tests will be conducted in the Pacific Ocean in accordance with a contract signed between the China Ocean Mineral Resources Research & Development Association and the International Seabed Authority (ISA).The submersible is scheduled to spend 47 days at sea, according to a statement from the Ministry of Science and Technology.A submersible differs from a submarine, as it typically depends on another vessel or facility for support.The Jiaolong, designed to reach a depth of 7,000 meters, completed 17 dives in the South China Sea between May 31 and July 18 last year, reaching 3,759 meters during its deepest dive.In the year since the submersible's last dive, program team members have made several technical improvements to the submersible and its support vessel, Jin said.Jin said the program team members are "very confident" about the test.He also stressed that the test will be carried out in accordance with the provisions of the United Nations Convention on the Law of the Sea as well as the ISA's rules and regulations.The Jiaolong is the world's first manned submersible designed to reach depths of 7,000 meters below sea level, according to Xu Qinan, the submersible's chief designer.

LOS ANGELES, Sept. 1 (Xinhua) -- Fisker Automotive, a Southern California electric car maker, announced on Thursday that it has signed a pact with BMW regarding the supply of engines and other related components for future models in a major tieup.The Anaheim, Orange County-based U.S. car maker will get a four-cylinder turbocharged engine, totalling 100,000 units every year, for its next generation of cars under a project code-named "Project Nina," the company said.The production of the model, a mass-market plug-in hybrid sedan, will begin at the end of 2012 and the sale is slated for 2013."The BMW engine was an obvious choice for us, as BMW is known for producing the best and most fuel efficient gasoline engines in the world," Fisker's CEO and Executive Design Director Henrik Fisker said. "We are very pleased to have signed this agreement with BMW."The firm did not disclose the terms.The U.S. automaker, founded in 2007, has produced electric vehicles with extended range (EVer). The first derivative of "Project Nina" will be a mid-sized premium sedan which utilizes the technology.The company's other products include Fisker Karma Sedan, a luxury sports car and the world's first premium electric plug-in hybrid.

CANBERRA, Aug. 31 (Xinhua) -- Australia's new chief medical officer, Professor Chris Baggoley, on Wednesday said Australian should not be worried about reports that a mutant strain of the deadly bird flu virus is spreading across Asia and beyond.On Monday, the UN's Food and Agriculture Organization (FAO) said a mutant strain of the deadly avian influenza virus was spreading in Asia and issued a warning that the bird flu could spread from poultry to humans. It urged "heightened readiness and surveillance" as the mutant strain posed "unpredictable risks to human health."But Professor Baggoley, who took office on Tuesday, said Australia is well served by strict testing and customs regimes, adding that avian migration patterns also mean infected birds are highly unlikely to arrive in Australia."I don't think we should be worried," Prof Baggoley told Sky News."The country and the Australian government is certainly vigilant in relation to bird flu."Prof. Baggoley insisted the H5N1 virus remains overwhelmingly a disease of birds and it is very uncommon for humans to catch it. It is also exceedingly rare for humans to spread it among themselves.He said that even if bird flu did arrive and affected humans, Australia is ready to respond to any new and significant developments in bird flu."Australia tests migratory wild birds, looking for H5N1, and has been doing this for some years and has never found it," he said."On the human side of things, Australia has been preparing for a possible outbreak of H5N1 since 2004."We've got the laboratory capacity to diagnose influenza; we've got arrangements in place for a rapid production of an H5N1 vaccine for humans, and we've got ready access to antivirals."There have been no outbreaks of bird flu in Australia to date.The UN said the latest human death from bird flu occurred earlier this month in Cambodia, which has registered eight cases of human infection this year, all of them fatal.H5N1 has infected 565 people since it first appeared in 2003, killing 331.

BEIJING, August 1 (Xinhuanet) -- U.S. researchers have discovered a genetic mutation unique to African Americans that links to heighten their asthma risk, according to reports published on Sunday in the journal Nature Genetics.Researchers of University of Chicago found a gene, PYHIN1, and its variations may account for asthma risk in people of African descent. In their study, they examined data from nine previous genome-wide association studies, totaling more than 2 million single nucleotide polymorphisms in 3,246 asthma patients and 3,385 control patient, 1,702 patient-parent groupings, 355 family-based cases, and 468 family-based controls.In the study, the researchers found that between 26 percent and 29 percent of people of African-descent carried at least one copy of the gene.  In particular, one polymorphism of the gene was 34 percent more likely found in African-American and Afro-Caribbean people with asthma.  More interestingly, this variant of the gene is rarely found in Caucasian people and in Latino population, it was found in less than 5 percent.This new variant is part of a family of genes linked with the body's response to viral infections. It is thought to be involved in interferon signaling, which could influence the occurrence of asthma.The team stressed that each gene variant on its own plays only a small role in increasing asthma risk, but that risk could be multiplied when combined with other risk genes and with environmental factors.