济南牛蒡茶可以降尿酸吗(山东严重痛风病的输液治疗) (今日更新中)

WELLINGTON, June 7 (Xinhua) -- Scientists in New Zealand have developed a new drug to fight previously untreatable hypoxic cancer tumors, which form in areas of the body starved of oxygen.The researchers at the Auckland University have entered an agreement for the clinical development of CEN-209, which was developed over 10 years of research, said a statement from the university Tuesday.CEN-209 was designed to enhance the effectiveness of radiotherapy and chemotherapy in solid hypoxic tumors, which were resistant to standard cancer therapies, said the statement. In lung cancer patients for example, about half of tumors had hypoxic regions.The new drug worked by damaging the DNA of hypoxic cancer cells, while leaving normal, healthy tissues alone.CEN-209 was designed and created by researchers at the university's Auckland Cancer Society Research Centre (ACSRC), using computer models of drug transport within tumors to accurately predict the anti-tumor activity of the drugs."Our computer models of drug transport developed in-house allowed the synthetic chemists to test their design theories and considerably shortened the discovery process," said Associate Professor Michael Hay, who led the ACSRC research chemists."CEN-209 improves markedly on previous agents in this class in terms of its ability to penetrate tumors, and this is reflected by its improved activity in the laboratory, when combined with long or short courses of radiotherapy," said researcher Professor Bill Wilson.Under the agreement between the university's Auckland UniServices Ltd. and California-based Centella Therapeutics, Inc., a subsidiary of Varian Medical Systems, Inc., Centella will have exclusive rights to CEN-209, which it will develop and trial with Cancer Research UK.The work on CEN-209 is the culmination of a program initiated with funding from the U.S. National Cancer Institute, and more recently from the Maurice Wilkins Centre for Biodiscovery. Ongoing preclinical research on CEN-209 and a backup compound was funded by grants from the Auckland Medical Research Foundation, Genesis Oncology Trust and Health Research Council of New Zealand, said the statement.

SAN FRANCISCO, Aug. 22 (Xinhua) -- Hewlett-Packard's tablet TouchPad has become the hottest consumer electronics device on Monday after HP launched a fire sale for the discontinued product line over the weekend."It was like a stampede and they went quick," a Best Buy employee in San Diego told technology news website Betanews, describing the line waiting to buy TouchPad. He said the discontinued tablet was sold out five minutes after the store opened.The device is also getting snatched up at a lighting speed online and increasingly difficult to find.The TouchPad is no longer listed on Bestbuy.com in the United States, which last week was reportedly sitting on 270,000 unsold TouchPads and was hoping HP would buy them back.On Amazon, TouchPad tops the world's largest online retailer's electronics sales list, beating out the electronic reader Kindle and iPod touch.Last Thursday, HP announced that it will completely shut down its webOS operations, specifically the TouchPad tablet and webOS phones.The 16 GB TouchPad currently is only 99.99 U.S. dollars, compared to the original price of 399 dollars. The 32 GB model, which used to be 485.99 dollars, is now priced at 149.99 dollars.

BERLIN, June 10 (Xinhua) -- The German state of North Rhine-Westphalia said Friday it detected for the first time the deadly E.coli strain o104 on bean sprouts, after they were named the source of the outbreak by Germany's national disease control centre. "According to our knowledge, the bean sprouts are coming from the recent suspicious farm in Bienenbuettel in the state of Lower Saxony," said Johannes Remmel, consumer protection minister.The sprouts were found in an opened package which had been left in a dustbin of a family, living near the city of Bonn. Two of the family members had eaten the sprouts and contracted the E. coli infection in mid-May, he said.A notice warning consumers not to eat raw bean sprouts is seen at a market in Berlin, capital of Germany, June 10, 2011. The German authority said on Friday bean sprouts were probably the source of the E. coli outbreak, which has killed 30 people and infected about 3,000 around the world. Reinhard Burger, president of the Robert Koch Institute (RKI), Germany's national disease control center said the Robert Koch Institute was lifting its warning against eating cucumbers, tomatoes and lettuce but keeping the warning in place for the sprouts."The discovery confirms our current warning against the consumption of bean sprouts. It is therefore becoming increasingly more likely that bean sprouts are the source of the E.coli infections," Remmel said.The news came after Reinhard Burger, President of the Robert Koch Institute (RKI), said in the morning bean sprouts were the source for the outbreak based on a epidemiological investigation."People who ate (bean) sprouts were found nine times more likely to have bloody diarrhoea or other signs of E. coli infection than those who did not," he saidHowever, no sample tests had found the o104 strain on bean sprouts when he announced that conclusion. Laboratory tests have shown Germany made mistakes in identifying the outbreak source on two previous occasions.At the same time, RKI lifted the warning against cucumbers, tomatoes and lettuce."Enjoy lettuce, cucumbers and tomatoes. They are healthy for you," said Andreas Hensel, president of the German Federal Risk Assessment Institute (BFR), which is a co-leader in the action against E. coli.In the city of Hamburg, an epicentre for the disease, farmers protested in the city centre by offering tons of lettuce and cucumbers for free to anyone who wanted them when the news was announced. Suddenly, pedestrians turned from reluctant to eager takers, reported local news agency DPA.

BEIJING, June 27 (Xinhuanet) -- A "lean gene" that helps people keep slim was found closely related to diabetes and heart disease, according to a study pulished in Nature Genetics journal on Sunday.The scientists from Britain’s Medical Research Council Epidemiology Unit conducted the study.After comparing the genetic codes of more than 75,000 people, a gene called IRS1 was spotted as the decisive factor to lower the fat level in human body.But in further study, IRS1 was found putting people to a 20 percent higher risk of developing heart disease and type-2 diabetes.The study warned that people who appear slim and fit might be also at risk.But scientists suggest that the study results did not change the general message for most people."While this study pointed to genes as one factor in determining the risk of developing these conditions, it was important to remember that lifestyle factors like diet, exercise, cutting out smoking and maintaining a healthy weight also play a vital role in reducing the risk," said Ruth Loos, who led the study.

WASHINGTON, June 14 (Xinhua) -- Eating a low-carbohydrate, high- protein diet may reduce the risk of cancer and slow the growth of tumors already present, according to a study published Tuesday in Cancer Research, a journal of the American Association for Cancer Research.The study was conducted in mice, but the scientists involved agree that the strong biological findings are definitive enough that an effect in humans can be considered."This shows that something as simple as a change in diet can have an impact on cancer risk," said lead researcher Gerald Krystal, a scientist at the British Columbia Cancer Research Center.Krystal and his colleagues implanted various strains of mice with human tumor cells or with mouse tumor cells and assigned them to one of two diets. The first diet, a typical Western diet, contained about 55 percent carbohydrate, 23 percent protein and 22 percent fat. The second, which is somewhat like a South Beach diet but higher in protein, contained 15 percent carbohydrate, 58 percent protein and 26 percent fat. They found that the tumor cells grew consistently slower on the second diet.As well, mice genetically predisposed to breast cancer were put on these two diets and almost half of them on the Western diet developed breast cancer within their first year of life while none on the low-carbohydrate, high-protein diet did. Interestingly, only one on the Western diet reached a normal life span ( approximately 2 years), with 70 percent of them dying from cancer while only 30 percent of those on the low-carbohydrate diet developed cancer and more than half these mice reached or exceeded their normal life span.Krystal and colleagues also tested the effect of an mTOR inhibitor, which inhibits cell growth, and a COX-2 inhibitor, which reduces inflammation, on tumor development, and found these agents had an additive effect in the mice fed the low-carbohydrate, high-protein diet.When asked to speculate on the biological mechanism, Krystal said that tumor cells, unlike normal cells, need significantly more glucose to grow and thrive. Restricting carbohydrate intake can significantly limit blood glucose and insulin, a hormone that has been shown in many independent studies to promote tumor growth in both humans and mice.Furthermore, a low-carbohydrate, high-protein diet has the potential to both boost the ability of the immune system to kill cancer cells and prevent obesity, which leads to chronic inflammation and cancer.

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