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WASHINGTON, June 21 (Xinhua) -- A new study from Harvard School of Public Health (HSPH) and University of California, San Francisco, researchers suggests that men with prostate cancer who smoke increase their risk of prostate cancer recurrence and of dying from the disease. The study will be published Wednesday in the Journal of the American Medical Association."In our study, we found similar results for both prostate cancer recurrence and prostate cancer mortality," said Stacey Kenfield, lead author and a research associate in the HSPH Department of Epidemiology. "These data taken together provide further support that smoking may increase risk of prostate cancer progression."Kenfield and her colleagues conducted a prospective observational study of 5,366 men diagnosed with prostate cancer between 1986 and 2006 in the Health Professionals Follow-Up Study. The researchers documented 1,630 deaths, 524 (32 percent) due to prostate cancer, 416 (26 percent) due to cardiovascular disease, and 878 prostate cancer recurrences.The researchers found that men with prostate cancer who were current smokers had a 61 percent increased risk of dying from prostate cancer, and a 61 percent higher risk of recurrence compared with men who never smoked. Smoking was associated with a more aggressive disease at diagnosis, defined as a higher clinical stage or Gleason grade (a measure of prostate cancer severity). However, among men with non-metastatic disease at diagnosis, current smokers had an 80 percent increased risk of dying from prostate cancer.Compared with current smokers, men with prostate cancer who had quit smoking for 10 or more years, or who had quit for less than 10 years but smoked less than 20 pack-years before diagnosis, had prostate cancer mortality risk similar to men who had never smoked. Men who had quit smoking for less than 10 years and had smoked 20 or more pack-years had risks similar to current smokers."These data are exciting because there are few known ways for a man to reduce his risk of dying from prostate cancer," said senior author Edward Giovannucci, professor of nutrition and epidemiology at HSPH. "For smokers, quitting can impact their risk of dying from prostate cancer. This is another reason to not smoke."Prostate cancer is the most frequently diagnosed form of cancer diagnosed in the United States and the second leading cause of cancer death among U.S. men, affecting one in six men during their lifetime. More than two million men in the U.S. and 16 million men worldwide are prostate cancer survivors.

TEHRAN, June 15 (Xinhua) -- Iran puts the Rasad (surveillance) satellite in the orbit on Wednesday, the state IRIB TV website reported.The Rasad satellite will render images to the country, said the report.All the procedure of designing, construction, testing and preparations to launch the satellite have been conducted domestically by Iranian experts, according to IRIB.The Rasad, weighs 15.3 kilograms and has been designed to be launched at 260 kilometers above the earth, said IRIB, adding that it will circle the earth 15 times in 24 hours.

HONG KONG, July 19 (Xinhua) -- A gene controlled by male hormone was critical for the growth of liver tumor, leading to a higher incidence of the disease in men than in women, according to the Chinese University of Hong Kong which made the results public on Tuesday.There is a significant gender bias in liver cancer incidence. In Hong Kong, men are three times more likely to develop liver cancer than women. Such phenomenon prompted researchers from the university to carry out a genomic location analysis in liver cancer cells starting from 2008.The researchers discovered, out of 17,000 human genes, that a gene called Cell Cycle-Related Kinase (CCRK) was directly controlled by androgen receptor. They further found that CCRK was critical for the induction of signaling pathway leading to abnormal liver cell growth and tumor formation.The results also reveal that 70 percent of patients with liver cancer were found to produce high levels of CCRK. More importantly, these patients were more likely to have late-stage cancers and lived shorter than those with low CCRK level.Using mouse models, the researchers found that either lowering the level or blocking the signaling pathway of CCRK in liver cancer cells could significantly reduce the tumor growth rate.The study has validated that CCRK is an important gene that has potential to cause liver cancer and the gene can be tested as a new targeted therapy in future.Vice-Chancellor of the university Joseph Sung, who is also team leader of the research project, said the results explained why men have a higher risk of liver cancer than women and helped push ahead with the development of a novel treatment of the disease.The above study results were published online in July in Journal of Clinical Investigation.

BEIJING, June 14 (Xinhuanet) -- A biographical comic book of Apple's CEO Steve Jobs will hit the shelves in August, according to media reports Tuesday. Titled "Steve Jobs: Co-Founder of Apple", the 32-page comic book, which details the life and career of Jobs, will be published by Bluewater Productions Inc. "His innovations command front page news, speculation of his health affects the stock market. Not bad for a college dropout," Bluewater president Darren Davis said in a statement. "His story, and that of Apple, is epic." Apple CEO Steve Jobs.The book, priced at 3.99 U.S. dollars, was written by C.W. Cooke and drawn by Chris Schmidt. The publisher's intention to make Jobs as its subject came from the success of the comic biography of Facebook founder Mark Zuckerberg. "There are definitely some similarities between Zuckerberg and Jobs. It takes a certain kind of drive and a certain kind of genius to move society the way they have," said C.W. Cooke. In addition, the first authorized biography of Jobs, "iSteve: The Book of Jobs", will be released on March 6, 2012.

来源：资阳报