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BEIJING, Sept. 2 (Xinhuanet) -- Firefighters who worked in the wreckage of the World Trade Center in 2001 were 19 percent more likely to develop cancer than those who were not there, according to a study.The study, published Thursday in the British medical journal The Lancet, surveyed cancer occurrence in nearly 10,000 male firefighters in the seven years after Sept. 11, 2001. (There were too few women to create a meaningful sample size.)The 9/11 attacks occurred on Tuesday, Sept. 11, 2001. As a result, the World Trade Center collapsed, and nearly 3,000 Americans killed. Among the 2,753 victims killed in the World Trade Center were 343 firefighters.There were 263 cancer cases in the exposed population, showing a cancer rate 19 percent higher than that of the group not exposed.The study indicated that cancers like melanoma, non-Hodgkin’s lymphoma, thyroid and prostate cancer occurred more frequently among exposed firefighters than in the general population. But occurrences of lung cancer did not increase.The findings “provide information that there may be a significant cancer risk for these people”, said Dr. James Melius, the administrator of the New York State Laborers’ Health and Safety Trust Fund and one of the peer reviewers of the study.But the results were far from conclusive. “This is not an epidemic,” said Dr. David J. Prezant, a lead researcher and the chief medical officer for the New York Fire Department.

SAN FRANCISCO, June 15 (Xinhua) -- Facebook has hired Joe Lockhart, who was former U.S. President Bill Clinton's press secretary during the last two years of his second term, as the social network company's vice president of global communications, U.S. media reported on Wednesday.Lockhart, 51, will report to Elliot Schrage, Facebook's current vice president of global communications, marketing and public policy, The Wall Street Journal said in a report."His experience building and running a press office at the White House gives him particular appreciation for the demands of a global 24-hour news cycle and the challenges of responding effectively to intense scrutiny," Schrage said of Lockhart's arrival.Lockhart will start at Facebook on July 15 and will move from Washington D.C. to Facebook's headquarters in California.Lockhart is the latest Washington insider to join Facebook as the world's largest social networking site is facing intense scrutiny for its privacy practices and growing global presence, The Wall Street Journal pointed out.Last year, Facebook also hired White House economic adviser Marne Levine to serve as its vice president of global public policy.More recently, Facebook hired former Bush administration officials Joel Kaplan as its vice president for U.S. public policy, and Myriah Jordan, who will become a policy manager focusing on congressional relations.

BERLIN, June 10 (Xinhua) -- The German state of North Rhine-Westphalia said Friday it detected for the first time the deadly E.coli strain o104 on bean sprouts, after they were named the source of the outbreak by Germany's national disease control centre. "According to our knowledge, the bean sprouts are coming from the recent suspicious farm in Bienenbuettel in the state of Lower Saxony," said Johannes Remmel, consumer protection minister.The sprouts were found in an opened package which had been left in a dustbin of a family, living near the city of Bonn. Two of the family members had eaten the sprouts and contracted the E. coli infection in mid-May, he said.A notice warning consumers not to eat raw bean sprouts is seen at a market in Berlin, capital of Germany, June 10, 2011. The German authority said on Friday bean sprouts were probably the source of the E. coli outbreak, which has killed 30 people and infected about 3,000 around the world. Reinhard Burger, president of the Robert Koch Institute (RKI), Germany's national disease control center said the Robert Koch Institute was lifting its warning against eating cucumbers, tomatoes and lettuce but keeping the warning in place for the sprouts."The discovery confirms our current warning against the consumption of bean sprouts. It is therefore becoming increasingly more likely that bean sprouts are the source of the E.coli infections," Remmel said.The news came after Reinhard Burger, President of the Robert Koch Institute (RKI), said in the morning bean sprouts were the source for the outbreak based on a epidemiological investigation."People who ate (bean) sprouts were found nine times more likely to have bloody diarrhoea or other signs of E. coli infection than those who did not," he saidHowever, no sample tests had found the o104 strain on bean sprouts when he announced that conclusion. Laboratory tests have shown Germany made mistakes in identifying the outbreak source on two previous occasions.At the same time, RKI lifted the warning against cucumbers, tomatoes and lettuce."Enjoy lettuce, cucumbers and tomatoes. They are healthy for you," said Andreas Hensel, president of the German Federal Risk Assessment Institute (BFR), which is a co-leader in the action against E. coli.In the city of Hamburg, an epicentre for the disease, farmers protested in the city centre by offering tons of lettuce and cucumbers for free to anyone who wanted them when the news was announced. Suddenly, pedestrians turned from reluctant to eager takers, reported local news agency DPA.

LOS ANGELES, July 18 (Xinhua) -- At least 70 genetic mutations may be involved in the formation of colon cancer, far more than scientists previously thought, a new study suggests.The study by researchers at University of Texas (UT) Southwestern Medical Center contradicts previous thinking that only a few mutated genes may play a role in the development of colon cancer."The ways we've been treating patients up to now is to just go after one target when we should be going after three to four different pathways simultaneously," said Dr. Jerry W. Shay, vice chairman and professor of cell biology at UT Southwestern.The new study identified 65 candidate genes and at least five passenger genes whose mutations play significant roles in cancer development. Inactivating the function of any of these tumor- suppressing genes led to a key step in cancer development called anchorage-independent growth, meaning cells piled up on top of each other rather than aligning neatly.According to previous studies, there were 151 candidate genes and that mutations in just eight to 15 of them would lead to cancer. There were 700 other genes classified as passenger genes whose mutations were incidental to cancer growth.Current cancer treatments target just one or two known cancer- driver genes. While patients may get transient tumor burden reduction, almost universally tumor growth returns."Those numbers are dead wrong," Dr. Shay said, suggesting a new approach to colon cancer treatments targeting multiple genes and pathways simultaneously.The next step is further research to classify more accurately which genes drive cancer and which are merely passengers, the researchers said.Study findings were published in the July 2011 Cancer Research (Priority Reports).

LOS ANGELES, July 18 (Xinhua) -- At least 70 genetic mutations may be involved in the formation of colon cancer, far more than scientists previously thought, a new study suggests.The study by researchers at University of Texas (UT) Southwestern Medical Center contradicts previous thinking that only a few mutated genes may play a role in the development of colon cancer."The ways we've been treating patients up to now is to just go after one target when we should be going after three to four different pathways simultaneously," said Dr. Jerry W. Shay, vice chairman and professor of cell biology at UT Southwestern.The new study identified 65 candidate genes and at least five passenger genes whose mutations play significant roles in cancer development. Inactivating the function of any of these tumor- suppressing genes led to a key step in cancer development called anchorage-independent growth, meaning cells piled up on top of each other rather than aligning neatly.According to previous studies, there were 151 candidate genes and that mutations in just eight to 15 of them would lead to cancer. There were 700 other genes classified as passenger genes whose mutations were incidental to cancer growth.Current cancer treatments target just one or two known cancer- driver genes. While patients may get transient tumor burden reduction, almost universally tumor growth returns."Those numbers are dead wrong," Dr. Shay said, suggesting a new approach to colon cancer treatments targeting multiple genes and pathways simultaneously.The next step is further research to classify more accurately which genes drive cancer and which are merely passengers, the researchers said.Study findings were published in the July 2011 Cancer Research (Priority Reports).

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