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WASHINGTON, June 16 (Xinhua) -- Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage. New research by a team of scientists at the University of Rochester has unveiled an important new mechanism that allows cells to recognize when they are under stress and prime the DNA repair machinery to respond to the threat of damage.Their findings will be published Friday in journal Science. Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage.The scientists, led by biologists Vera Gorbunova and Andrei Seluanov, focused on the most dangerous type of DNA damage -- double strand breaks. Unrepaired, this type of damage can lead to premature aging and cancer. They studied how oxidative stress affects efficiency of DNA repair. Oxidative stress occurs when the body is unable to neutralize the highly-reactive molecules, which are typically produced during routine cellular activities.The research team found that human cells undergoing oxidative stress synthesized more of a protein called SIRT6. By increasing SIRT6 levels, cells were able to stimulate their ability to repair double strand breaks. When the cells were treated with a drug that inactivated SIRT6, DNA repair came to a halt, thus confirming the role of SIRT6 in DNA repair. Gorbunova notes that the SIRT6 protein is structurally related to another protein, SIR2, which has been shown to extend lifespan in multiple model organisms."SIRT6 also affects DNA repair when there is no oxidative stress," explains Gorbunova. "It's just that the effect is magnified when the cells are challenged with even small amounts of oxidative stress."SIRT6 allows the cells to be economical with their resources, priming the repair enzymes only when there is damage that needs to be repaired. Thus SIRT6 may be a master regulator that coordinates stress and DNA repair activities, according to Gorbunova.

XICHANG, Sichuan, July 12 (Xinhua) -- China blasted off a new data relay satellite "Tianlian I-02" on Monday at the Xichang Satellite Launch Center in southwest Sichuan Province .The satellite was launched on a Long March-3C carrier rocket at 11:41 p.m. (Beijing Time), said sources with the center.Developed by the China Academy of Space Technology under the China Aerospace Science and Technology Corporation, the satellite is the country's second data relay satellite.China launched its first data relay satellite "Tianlian I-01" on April 25, 2008.The two satellites will form a network to offer data relay and measurement and control service for China's spacecrafts and planned space stations, according to the center.They will also be used to help perform the nation's first space docking, scheduled for the second half of 2011.China plans to launch Tiangong-1 and Shenzhou-8 spacecraft in the latter half of this year, and they will perform the nation's first space docking.Monday's launch is the 140th mission of China's Long March series of rockets.

YANGON, July 10 (Xinhua) -- Myanmar is projecting to build the first-ever liver transplant hospital in line with the international standard, the local weekly Voice reported Sunday.With the technological help of the Changi General Hospital of Singapore, a 40-million-U.S dollar worth private hospital has started building since late last month.The hospital will offer services for the patients living with heart and kidney diseases and for protection from being affected Methicillin-Resistant Staphylococcus Aureus (MRSA), the report said.In Myanmar, liver transplant will cost about 20 million Kyats ( 25,000 U.S. dollars), lesser than other countries, the report added.Myanmar experts carried out successful liver-transplant operation in 2004 for the first time and in 2009 for the second time.

BRUSSELS, May 31 (Xinhua) -- A deadly outbreak of E.coli linked to contaminated cucumbers is sending Europe on high alert, as death toll from the disease climbed to 16 till Tuesday.So far, 15 people have reportedly died and more than 300 were seriously ill from infections linked to tainted vegetables in Germany. Experts said the numbers are expected to increase in the coming week."We hope the number of cases will go down but we fear it will worsen," said Oliver Grieve, spokesperson with the University Medical Center Schleswig where most German victims are being treated.The German government has identified the pathogen as hemolytic-uremic syndrome (HUS), a serious complication of a type of E. coli known as Shiga toxin-producing E.coli (STEC). The bacteria could damage blood cells and kidneys, and hence cause deaths.A woman in her 50s was confirmed to be the first fatal victim out of Germany by a local hospital on Tuesday. Officials at the hospital in Boras, southwestern Sweden, said the woman, who had recently traveled to Germany, was admitted on May 29.Swedish health authorities already said on Sunday that 36 people, all linked to travel in northern Germany, had been confirmed to have infected with E.coli, whereas cases were also reported in France, the Netherlands, Denmark and Britain.It was the second food scare for European consumers this year. In January, highly toxic dioxin was found in eggs, poultry and pork products across Germany.