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BEIJING, Sept. 27 (Xinhuanet) -- Last-minute preparations for the launch of the Tiangong-1 spacecraft began at the Jiuquan Satellite Launch Center on Monday, meaning the vehicle can soon embark on a mission that will eventually have it dock with a spaceship, according to the center.That feat, if carried out successfully, will mark the completion of China's first rendezvous and docking mission.At the launch site on Monday afternoon, crews were putting in place pipes and cables that will be used to inject fuel into the rocket that will carry Tiangong-1 into outer space.Without complications from the weather or other causes, the Long March II-F rocket will lift off from the launch center on Thursday or Friday, taking the Tiangong-1 with it, according to the Xinhua News Agency.Despite the preparations, space experts said carrying out the plan still comes with many risks. They explained that the spacecraft and much of the other equipment being used is new and has not been tried on an actual mission."Tiangong-1 is a brand new spacecraft designed by China and is bigger and heavier than the Shenzhou spaceships China had developed as a means of transporting astronauts from the Earth to space," said Yang Hong, chief designer of Tiangong-1.In a vertical position, the Tiangong-1 looks like a cigar standing 10.4 meter tall, a height equal to that of a three-story building. It weighs 8.5 tons and has a maximum diameter of 3.35 meters, a dimension shared by its launch vehicle, he said.In comparison, the Shenzhou spaceship stands shorter, at nearly 9 meters, is slimmer, having a diameter of less than 3 meters, and weighs less.In another difference, the Tiangong-1 is composed of two modules rather than the three that had made up the Shenzhou spaceship. Of the Tiangong-1's two primary components, one is an experimental module that contains a place that astronauts can live and work in on future missions. It is also equipped with a docking port.The other chief component, a resource module, will provide the craft with power.Astronauts on Tiangong-1 will have 15 cubic meters of space to move in, "much more than they had in the Shenzhou spaceship", Yang said.Inside the spacecraft are two sleeping sections with adjustable lighting systems, exercise equipment, entertainment systems and visual communication devices, he said.Hou Xiangyang, who helped design Tiangong-1, said: "The sleeping section is big enough for a 1.8-meter-tall man to sleep in. An astronaut can adjust the light as he likes."In space, Tiangong-1 will fly in a horizontal position. A paint scheme inside will help the astronauts aboard maintain their sense of direction; the module's inner walls will be in two colors, one commonly associated with the sky and one with the ground."This will help astronauts avoid feeling as if they are standing upside down in the microgravity environment," he said.Yang said the Tiangong-1 spacecraft is expected to stay in orbit for two years and rendezvous and dock with three different spaceships. Beyond the Shenzhou VIII, ships named Shenzhou IX and Shenzhou X will embark on similar missions; at least one of the two is to be manned.Before astronauts climb on board Tiangong-1, the conditions inside its experimental module will be adjusted to ensure they can live in an environment that contains enough oxygen, moisture and heat to be safe.Tiangong-1 was originally scheduled to be launched into a low orbit around the Earth between Sept 27 and 30. The earlier days were removed as possible launch dates, though, after forecasters predicted a cold air mass would move into the area containing the Jiuquan Satellite Launch Center.To ensure the spacecraft and Long March II-F carrier rocket are ready for launch, a full ground simulation was conducted on Sunday afternoon.

WASHINGTON, Aug. 4 (Xinhua) -- U.S. scientists have found two gene mutations occurring in oligodendrogliomas, the second-most common form of brain cancer, according to a study to be published Friday in journal Science.For years scientists have been looking for the primary cancer genes involved in oligodendrogliomas evolvement. Scientists know the two chromosomes held the probable mutations, but the particular gene information remains unclear.Now scientists at Duke University Medical Center and Johns Hopkins University have discovered the most likely genetic mutations that researchers have been hunting for on chromosomes 1 and 19.The genes they identified, CIC or FUBP1, are tumor suppressor genes. The cancer-related pathways that involve these genes could become targets for future treatments, said Hai Yan, a Duke associate professor of pathology and co-corresponding author of the study.The researchers found CIC on chromosome 19 and FUBP1 on chromosome 1 based on an initial study of seven oligodendrogliomas. They found six mutations and two mutations, respectively, in the seven tumors. Further study of 27 more of these tumors showed that there were 12 and three mutations of CIC and FUBP1, respectively. The two genes were rarely mutated in other types of cancers, indicating that they are oligodendroglioma-specific genes.These genes were difficult to find until the technology improved, said Yan."The team used whole genome sequencing technology so that no genes would be excluded, and we found to our surprise that one gene, on chromosome 19, was mutated in six out of the seven initial tumor specimens we sequenced," Yan said. "A mutation frequency of 85 percent is very high."The finding of two additional new genes involved in oligodendrogliomas increases the chances for an effective combination drug therapy for the tumor, Yan said. He envisions a combination cocktail of drugs similar to the combination-drug treatments taken by HIV patients that would target different pathways involved in cancer, and assist both in reducing the chance of relapsing and increasing odds of success.

WASHINGTON, June 21 (Xinhua) -- A new study from Harvard School of Public Health (HSPH) and University of California, San Francisco, researchers suggests that men with prostate cancer who smoke increase their risk of prostate cancer recurrence and of dying from the disease. The study will be published Wednesday in the Journal of the American Medical Association."In our study, we found similar results for both prostate cancer recurrence and prostate cancer mortality," said Stacey Kenfield, lead author and a research associate in the HSPH Department of Epidemiology. "These data taken together provide further support that smoking may increase risk of prostate cancer progression."Kenfield and her colleagues conducted a prospective observational study of 5,366 men diagnosed with prostate cancer between 1986 and 2006 in the Health Professionals Follow-Up Study. The researchers documented 1,630 deaths, 524 (32 percent) due to prostate cancer, 416 (26 percent) due to cardiovascular disease, and 878 prostate cancer recurrences.The researchers found that men with prostate cancer who were current smokers had a 61 percent increased risk of dying from prostate cancer, and a 61 percent higher risk of recurrence compared with men who never smoked. Smoking was associated with a more aggressive disease at diagnosis, defined as a higher clinical stage or Gleason grade (a measure of prostate cancer severity). However, among men with non-metastatic disease at diagnosis, current smokers had an 80 percent increased risk of dying from prostate cancer.Compared with current smokers, men with prostate cancer who had quit smoking for 10 or more years, or who had quit for less than 10 years but smoked less than 20 pack-years before diagnosis, had prostate cancer mortality risk similar to men who had never smoked. Men who had quit smoking for less than 10 years and had smoked 20 or more pack-years had risks similar to current smokers."These data are exciting because there are few known ways for a man to reduce his risk of dying from prostate cancer," said senior author Edward Giovannucci, professor of nutrition and epidemiology at HSPH. "For smokers, quitting can impact their risk of dying from prostate cancer. This is another reason to not smoke."Prostate cancer is the most frequently diagnosed form of cancer diagnosed in the United States and the second leading cause of cancer death among U.S. men, affecting one in six men during their lifetime. More than two million men in the U.S. and 16 million men worldwide are prostate cancer survivors.

HELSINKI, July 4 (Xinhua) -- A new Finnish research shows that sleeplessness may be hereditary, and insomniacs are more likely to die earlier than people with healthy sleep patterns.The research is the first to link insomnia with mortality risk, Finnish media reported on Monday.The research is conducted by the Institute of Occupational Health in collaboration with the University of Helsinki and the Finnish National Institute for Health and WelfareIn a large-scale twin study, the Finnish researchers followed the health status of 12,500 adult twin pairs during the years from 1990 to 2009. Twenty percent of the participants were suffering from sleeplessness symptoms, including difficulty in initiating sleep, nocturnal awakening and non-restorative sleep.The study found out that compared with unidentical twins, identical twins were more likely to suffer from similar insomnia symptoms. This finding indicates that genetic factors play a role in the formation of insomnia.Moreover, the participants were divided into three groups, according to their sleep qualities. Out of the participants, 48 percent were good sleepers, 40 percent average sleepers and 12 percent poor sleepers. The search result shows that insomnia-related symptoms may increase mortality risk.In addition, compared with good sleepers, 7 percent of the women and 22 percent of the men who were average sleepers were more likely to die earlier; and poor sleepers were 1.5 times more likely to die earlier.According to the researchers, sleeplessness is a common health problem among working-age cohort. Chronic sleeplessness raises the risk of many illnesses and accidents, and thus weakens people's quality of life and ability to operate properly.The experts suggest that insomniacs should seek medical treatment in time, and chronic insomnia patients should be better treated with non-drug therapy.

BEIJING, Sept. 8 (Xinhuanet) -- Middle-aged white women drinking alcohol moderately are more likely to stay healthy than nondrinkers, according to a new study from Harvard.The study has followed about 14,000 mostly white women since 1976.As a result, compared with teetotalers, those who had 3 to 15 alcoholic drinks weekly in their late 50s were 28 percent higher of being free from physical disability, chronic illness, mental health problems, and cognitive decline at the age of 70.Even having just one or two drinks per week increased a woman's odds of good health by 11 percent.However, women should be aware that even moderate drinking has been linked to an increased risk of breast cancer, said Qi Sun, M.D., a lead author of the study and a nutrition researcher at the Harvard School of Public Health.These findings don't necessarily apply to men or to nonwhite women. But they reinforced the evidence for the health benefits of moderate drinking.

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