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SHANGHAI, July 19 (Xinhua) -- Baidu Inc., the largest Chinese search engine based in Beijing, said Tuesday it closed a deal that allows the company to provide download services of music from Universal Music, Warner Music and Sony Music.According to the deal signed between Baidu and One Stop China (OSC), a joint venture of the three music companies, Baidu will pay them on a per-play and per-download basis for all tracks delivered through its MP3 server, said a press release provided by Baidu.Users can have access to the authorized music free of charge, and Chinese songs, both in Mandarin and Cantonese, and some global content provided by the three music companies will be included in the catalogue.Baidu also launched a social music platform called "Baidu ting!" through which users can obtain and share some music-related information and content.Li Xinzhe, CFO of Baidu, described the deal as a "milestone" that can benefit not only Baidu and its partners but also music lovers and the privacy-stricken industry.Wang Bin, secretary general of Copyright Union of Internet Society of China, predicted that it would have a demonstrative effect on the Internet sector.In 2008, Universal, Warner and Sony Music sued Baidu for at least 63.5 million yuan (9.8 million U.S. dollars) in copyright infringement.

BEIJING, August 1 (Xinhuanet) -- U.S. researchers have discovered a genetic mutation unique to African Americans that links to heighten their asthma risk, according to reports published on Sunday in the journal Nature Genetics.Researchers of University of Chicago found a gene, PYHIN1, and its variations may account for asthma risk in people of African descent. In their study, they examined data from nine previous genome-wide association studies, totaling more than 2 million single nucleotide polymorphisms in 3,246 asthma patients and 3,385 control patient, 1,702 patient-parent groupings, 355 family-based cases, and 468 family-based controls.In the study, the researchers found that between 26 percent and 29 percent of people of African-descent carried at least one copy of the gene.  In particular, one polymorphism of the gene was 34 percent more likely found in African-American and Afro-Caribbean people with asthma.  More interestingly, this variant of the gene is rarely found in Caucasian people and in Latino population, it was found in less than 5 percent.This new variant is part of a family of genes linked with the body's response to viral infections. It is thought to be involved in interferon signaling, which could influence the occurrence of asthma.The team stressed that each gene variant on its own plays only a small role in increasing asthma risk, but that risk could be multiplied when combined with other risk genes and with environmental factors.

WASHINGTON, June 16 (Xinhua) -- Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage. New research by a team of scientists at the University of Rochester has unveiled an important new mechanism that allows cells to recognize when they are under stress and prime the DNA repair machinery to respond to the threat of damage.Their findings will be published Friday in journal Science. Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage.The scientists, led by biologists Vera Gorbunova and Andrei Seluanov, focused on the most dangerous type of DNA damage -- double strand breaks. Unrepaired, this type of damage can lead to premature aging and cancer. They studied how oxidative stress affects efficiency of DNA repair. Oxidative stress occurs when the body is unable to neutralize the highly-reactive molecules, which are typically produced during routine cellular activities.The research team found that human cells undergoing oxidative stress synthesized more of a protein called SIRT6. By increasing SIRT6 levels, cells were able to stimulate their ability to repair double strand breaks. When the cells were treated with a drug that inactivated SIRT6, DNA repair came to a halt, thus confirming the role of SIRT6 in DNA repair. Gorbunova notes that the SIRT6 protein is structurally related to another protein, SIR2, which has been shown to extend lifespan in multiple model organisms."SIRT6 also affects DNA repair when there is no oxidative stress," explains Gorbunova. "It's just that the effect is magnified when the cells are challenged with even small amounts of oxidative stress."SIRT6 allows the cells to be economical with their resources, priming the repair enzymes only when there is damage that needs to be repaired. Thus SIRT6 may be a master regulator that coordinates stress and DNA repair activities, according to Gorbunova.

LOS ANGELES, Sept. 1 (Xinhua) -- Fisker Automotive, a Southern California electric car maker, announced on Thursday that it has signed a pact with BMW regarding the supply of engines and other related components for future models in a major tieup.The Anaheim, Orange County-based U.S. car maker will get a four-cylinder turbocharged engine, totalling 100,000 units every year, for its next generation of cars under a project code-named "Project Nina," the company said.The production of the model, a mass-market plug-in hybrid sedan, will begin at the end of 2012 and the sale is slated for 2013."The BMW engine was an obvious choice for us, as BMW is known for producing the best and most fuel efficient gasoline engines in the world," Fisker's CEO and Executive Design Director Henrik Fisker said. "We are very pleased to have signed this agreement with BMW."The firm did not disclose the terms.The U.S. automaker, founded in 2007, has produced electric vehicles with extended range (EVer). The first derivative of "Project Nina" will be a mid-sized premium sedan which utilizes the technology.The company's other products include Fisker Karma Sedan, a luxury sports car and the world's first premium electric plug-in hybrid.

BEIJING, Aug. 30 (Xinhuanet) -- The search giant Google admits it sees the new Google+ social network as an "identity service" or platform on which it can build other products, according to media reports.Google chariman Eric Schmidt said Google isn't interested in changing its policies to accommodate users such as political dissidents or others who prefer to remain anonymous: If people want to remain anonymous, then they shouldn't use Google+."Fundamentally, [Google+] depends on people using their real names if they're going to build future products that leverage that information," NPR's Andy Carvin wrote in a post on Google+ as he paraphrased Schmidt's remarks.Critics say the move is harmful to political activists, victims of harassment and numerous other groups for whom using a real name online might pose a safety risk, according to CNN reports."Regarding people who are concerned about their safety, [Schmidt] said G+ is completely optional," Carvin wrote.Meanwhile, according to Carvin, Schmidt also said "the Internet would be better if we knew you were a real person rather than a dog or a fake person. Some people are just evil and we should be able to ID them and rank them downward."