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LOS ANGELES, June 14 (Xinhua) -- Rural elders are far more likely to be overweight or obese, physically inactive and food insecure than their suburban counterparts, three risk factors for heart disease, diabetes and repeated falls, a new study suggests."The countryside can have an isolating effect," said lead researcher Steven P. Wallace, deputy director of the Center for Health Policy Research at the University of California, Los Angeles. "When even a trip to the grocery store is a significant drive, seniors can become trapped in their houses."The researchers based their finding on analyzing the lifestyles and living conditions in California countryside.The study found that despite living in the countryside, where open space is plentiful and there is often significant agricultural production, California's more than half a million rural elders have higher rates of developing various health problems than their urban and suburban counterparts.These problems include:-- Older adults in rural areas are more often overweight or obese (61.3 percent) than their urban (57.3 percent) and suburban (54.0 percent) counterparts;-- Rural older adults do not get enough exercise;-- One in five rural elders do not participate in either moderate or vigorous physical activity in their leisure time;-- Rural and urban older adults are more likely to be food insecure; and-- One in five low-income older adults in rural settings report that they cannot consistently afford enough food to last the month, a rate is about twice that of low-income suburban adults.Approximately 710,000 Californians aged 65 and over live in the countryside -- almost one-fifth of all older adults in the state. Yet rural elders experience unique challenges to healthy living, including a lack of sidewalks, street lights, transportation services, access to healthy food outlets, parks, exercise facilities and health care sites. California's rural areas are also challenged by a dearth of physicians and other primary care providers, compelling many seniors to travel long distances to seek care, according to the study.The findings were published Tuesday on the website of the American Association for the Advancement of Science.

HELSINKI, July 4 (Xinhua) -- A new Finnish research shows that sleeplessness may be hereditary, and insomniacs are more likely to die earlier than people with healthy sleep patterns.The research is the first to link insomnia with mortality risk, Finnish media reported on Monday.The research is conducted by the Institute of Occupational Health in collaboration with the University of Helsinki and the Finnish National Institute for Health and WelfareIn a large-scale twin study, the Finnish researchers followed the health status of 12,500 adult twin pairs during the years from 1990 to 2009. Twenty percent of the participants were suffering from sleeplessness symptoms, including difficulty in initiating sleep, nocturnal awakening and non-restorative sleep.The study found out that compared with unidentical twins, identical twins were more likely to suffer from similar insomnia symptoms. This finding indicates that genetic factors play a role in the formation of insomnia.Moreover, the participants were divided into three groups, according to their sleep qualities. Out of the participants, 48 percent were good sleepers, 40 percent average sleepers and 12 percent poor sleepers. The search result shows that insomnia-related symptoms may increase mortality risk.In addition, compared with good sleepers, 7 percent of the women and 22 percent of the men who were average sleepers were more likely to die earlier; and poor sleepers were 1.5 times more likely to die earlier.According to the researchers, sleeplessness is a common health problem among working-age cohort. Chronic sleeplessness raises the risk of many illnesses and accidents, and thus weakens people's quality of life and ability to operate properly.The experts suggest that insomniacs should seek medical treatment in time, and chronic insomnia patients should be better treated with non-drug therapy.

WASHINGTON, Sept. 14 (Xinhua) -- The U.S. space agency NASA unveiled plans on Wednesday for a deep space rocket to carry astronauts to the moon, Mars and other destinations beyond the International Space Station.The Space Launch System, or SLS, will be designed to carry the Orion Multipurpose Crew Vehicle, as well as important cargo, equipment and science experiments to Earth's orbit and destinations beyond. Additionally, the SLS will serve as a backup for commercial and international partner transportation services to the International Space Station."This launch system will create good-paying American jobs, ensure continued U.S. leadership in space, and inspire millions around the world," NASA Administrator Charles Bolden said in a statement. "President Obama challenged us to be bold and dream big, and that's exactly what we are doing at NASA. While I was proud to fly on the space shuttle, kids today can now dream of one day walking on Mars."The SLS rocket will incorporate technological investments from the Space Shuttle program and the Constellation program in order to take advantage of proven hardware and cutting-edge tooling and manufacturing technology that will significantly reduce development and operations costs.The first developmental flight, or mission, is targeted for the end of 2017, according to NASA.

TEHRAN, June 15 (Xinhua) -- Iran puts the Rasad (surveillance) satellite in the orbit on Wednesday, the state IRIB TV website reported.The Rasad satellite will render images to the country, said the report.All the procedure of designing, construction, testing and preparations to launch the satellite have been conducted domestically by Iranian experts, according to IRIB.The Rasad, weighs 15.3 kilograms and has been designed to be launched at 260 kilometers above the earth, said IRIB, adding that it will circle the earth 15 times in 24 hours.

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."