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WASHINGTON, Oct. 13 (Xinhua) -- U.S. researchers have corrected sickle cell disease in adult laboratory mice that had been bred to have the inherited blood disorder by activating production of a special blood component, according to a study published online Thursday in the journal Science.Sickle cell disease results from an abnormality in hemoglobin, the protein found in red blood cells that is responsible for transporting oxygen throughout the body. People living with sickle cell disease have two copies of an altered gene that produces sickle hemoglobin instead of normal adult hemoglobin. Sickle hemoglobin changes shape after releasing its oxygen, causing the red blood cell to become stiff, misshapen and sticky, and slowing blood flow to tissues. This process damages organs and causes pain.The study tested a new approach to increasing the production of a third form of hemoglobin -- fetal hemoglobin. Production of fetal hemoglobin predominates before birth, but turns off thereafter as adult hemoglobin production takes over. People with sickle cell disease are unable to make normal adult hemoglobin, and instead make sickle hemoglobin starting in infancy.An elevated level of fetal hemoglobin within the red blood cell reduces the tendency of sickle hemoglobin to change the shape of red blood cells. Considerable research has shown that the drug hydroxyurea increases production of fetal hemoglobin and reduces the number of pain crises and other complications of sickle cell disease in adults and children. However, not all patients respond well to hydroxyurea, and adverse side effects are a concern.The current study explores a more targeted approach to increasing fetal hemoglobin production. It builds upon earlier studies that discovered a protein called BCL11A normally suppresses the production of fetal hemoglobin soon after birth. The researchers viewed the BCL11A protein as a target for therapy and decided to see what would happen if they blocked production of the protein.The paper details how the research team silenced the mouse gene that produces the BCL11A protein in mice with sickle cell disease. Silencing the gene turned off production of the BCL11A protein and allowed the adult mice to continue to produce fetal hemoglobin. It appears to have eliminated disease symptoms without affecting other aspects of blood production."This discovery provides an important new target for future therapies in people with sickle cell disease," said Susan Shurin, acting director of the U.S. National Heart, Lung, and Blood Institute, which co-funded the study. "More work is needed before it will be possible to test such therapies in people, but this study demonstrates that the approach works in principle."Approximately 100,000 Americans live with sickle cell disease. It is most prevalent in people of African, Hispanic, Mediterranean, and Middle Eastern descent. There is no widely available cure for sickle cell disease. Bone marrow transplants have cured some patients, but the treatment is not without risk and most patients do not have relatives who can donate compatible and healthy bone marrow to them.

WASHINGTON, Nov. 10 (Xinhua) -- Using its near-infrared vision to peer nine billion years back in time, NASA's Hubble Space Telescope has uncovered an extraordinary population of young dwarf galaxies brimming with star formation, the U.S. space agency announced on Thursday.While dwarf galaxies represent the most common type of galaxy in the universe, the rapid star-birth observed in these newly- found examples may force astronomers to reassess their understanding of the ways in which galaxies form.The galaxies are a hundred times less massive, on average, than the Milky Way, yet they churn out stars at such a furious pace that their stellar content would double in just 10 million years. By comparison, the Milky Way would take a thousand times longer to double its star population.The universe is estimated to be 13.7 billion years old, and these newly-discovered galaxies are extreme even for the young universe -- when most galaxies were forming stars at higher rates than they are today. Astronomers using Hubble's instruments could spot the galaxies because the radiation from young, hot stars has caused the oxygen in the gas surrounding them to light up like a bright neon sign."The galaxies have been there all along, but up until recently astronomers have been able only to survey tiny patches of sky at the sensitivities necessary to detect them," said Arjen van der Wel of the Max Planck Institute for Astronomy in Germany, lead author of a paper on the results to be published online on Nov. 14 in The Astrophysical Journal. "We weren't looking specifically for these galaxies, but they stood out because of their unusual colors. "The observations were part of the Cosmic Assembly Near-infrared Deep Extragalactic Legacy Survey (CANDELS), an ambitious three- year study to analyze the most distant galaxies in the universe. CANDELS is the first census of dwarf galaxies at such an early epoch."In addition to the images, Hubble has captured spectra that show us the oxygen in a handful of galaxies and confirmed their extreme star-forming nature," said co-author Amber Straughn at NASA's Goddard Space Flight Center. "Spectra are like fingerprints. They tell us the galaxies' chemical composition."The CANDELS team uncovered the 69 young dwarf galaxies in near- infrared images taken with Hubble's Wide Field Camera 3 and Advanced Camera for Surveys.The observations suggest that the newly-discovered galaxies were very common nine billion years ago. However, it is a mystery why the newly-found dwarf galaxies were making batches of stars at such a high rate.

OTTAWA, Oct. 3 (Xinhua) -- Many friends and colleagues of Canadian scientist Ralph Steinman reacted with shock when they learned on Monday that Steinman won the Nobel Prize for Medicine and Physiology three days after he died.Since 1974, Nobel Prizes are no longer awarded posthumously, but the Nobel Prize committee said that it had made its choice before Steinman's death.Many of Steinman's friends and colleagues said that they learned of Steinman's death at the same time that they learned of his Nobel Prize, which was awarded for a discovery Steinman made in 1973.Steinman, 68, discovered dendritic cells, which help regulate adaptive immunity, which purges invading microorganisms from the body. Dendritic cells activate T cells, which "remember" the DNA sequence of invading organisms and protect the body from later infections from the same disease."Their work has opened up new avenues for the development of prevention and therapy against infections, cancer and inflammatory disease," the citation said.Monday, the Nobel Committee defended its decision to award the prize to Steinman. "The decision to award the Nobel Prize to Ralph Steinman was made in good faith, based on the assumption that the Nobel Laureate was alive," the foundation said in a statement."The Nobel Foundation thus believes that what has occurred is more reminiscent of the example in the statutes concerning a person who has been named as a Nobel Laureate and has died before the actual Nobel Prize Award Ceremony."It is still unclear who will pick up Steinman's prize at the award ceremony later this year.Steinman, a cell biologist at Rockefeller University in New York City, died of pancreatic cancer on Friday. For more than four years, he had used his own immune therapy discoveries to extend his life."The news is bittersweet, as we also learned this morning from Ralph's family that he passed a few days ago," Rockefeller University President Marc Tessier-Lavigne said in a statement."We are all so touched that our father's many years of hard work are being recognized with a Nobel Prize," Steinman's daughter, Alexis, said in the statement. "He devoted his life to his work and his family, and he would be truly honored."Steinman's heirs will share the 1.5-million U.S. dollar prize with American genetics professor Bruce Beutler and French scientist Jules Hoffmann.Dr. Beutler is professor of genetics and immunology at The Scripps Research Institute in La Jolla, California. Dr. Hoffmann headed a research laboratory in Strasbourg, France, between 1974 and 2009 and served as president of the French National Academy of Sciences between 2007 and 2008."Ralph worked right up until last week," said Michel Nussenzweig, a collaborator of Steinman's at Rockefeller University. "His dream was to use his discovery to cure cancer and infectious diseases like HIV and tuberculosis. It's a dream that's pretty close."Steinman was born in 1943 in Montreal, Canada's second largest city, and studied chemistry and biology at McGill University in his hometown before receiving an MD from Harvard Medical School in Boston in 1968. He joined Rockefeller University in 1970 as a postdoctoral fellow."He was diagnosed with pancreatic cancer four years ago, and his life was extended using a dendritic-cell based immunotherapy of his own design," the university said in a statement.In a statement, Canadian Prime Minister Stephen Harper lauded the three winners of the Nobel for medicine and called the award " a fitting final tribute" to Steinman's life's work."Dr. Steinman shall be honored for all time with this achievement," Harper said. "Canadians will mourn his loss."

BEIJING, Nov. 17 (Xinhua) -- The Beijing municipal government Thursday launched a news release platform on Sina's weibo.com, China's popular Twitter-like microblogging service.Twenty municipal government agencies will post newly-unveiled policies and regulations, work developments, and information of news conferences on the joint microblog account, said Wang Hui, head of the Beijing municipal government's information office.The government departments will also timely answer hot topics and social concerns, Wang said.Many government departments have their own microblog accounts. The municipal public security bureau, for example, opened its microblog in August last year and now has over 1.8 million followers."I'm happy I can communicate directly with the government. And I hope my viewpoints and suggestions can get attention," said weibo user 1910060523.Many, however, question the move. "Is it really the case that everything can be said here and the government will read the comments and give responses?" said another user.Wang Hui said, "We are not just making a show. We shall be sincere to answer public's concerns over hot topics."

SAN FRANCISCO, Nov. 14 (Xinhua) -- Google is running a secret research lab in the San Francisco Bay Area, where the tech giant invests to experiment and invent what may be world-changing technologies for the future, U.S. media reported on Monday.According to The New York Times, at the lab dubbed Google X, engineers are working at some 100 projects from robots, smart refrigerators to Internet-enabled dinner plates and a "space elevator," a proposed non-rocket space launch structure.An unnamed Google engineer familiar with the lab told the newspaper that it was run as mysteriously as the CIA with two officers, a nondescript one for logistics on the company's Mountain View campus and one for robots in a secret location.Scientists working at the lab include many roboticists and electrical engineers hired from Microsoft, Nokia labs, Stanford, MIT, Carnegie Mellon and New York University. Google's co-founder Sergey Brin is said to be "deeply involved" in Google X.The lab is reportedly headed by Sebastian Thrun, one of the world's top robotics and artificial intelligence experts. He teaches computer science at Stanford University and invented the world's first self-driving car.A Google spokeswoman would not confirm the existence of the lab, but said Google likes to invest in speculative projects."While the possibilities are incredibly exciting, please do keep in mind that the sums involved are very small by comparison to the investments we make in our core businesses," she told The New York Times.