济南早泄治疗的药济南阴囊表皮上有小疙瘩,济南治疗早泄阳痿最好的药,济南现在割包皮痛吗,济南看阳痿哪家医院正规,济南治疗早泄用哪种中药好,济南阳痿自治疗,济南射精一分钟怎么回事

SAN FRANCISCO, Sept. 2 (Xinhua) -- A late-season potato blight has been detected on farms in Alaska, only the fourth time the disease appear in the U.S. state since 1995, local media reported on Friday.The disease was found this week in the fields of potato producers in cities of Delta Junction and Palmer, according to the Fairbanks Daily News-Miner, Alaska's second largest daily newspaper.Farmers in both fields are working to prevent the spread of the blight, a fungus-like disease that can kill plants in the field or cause potatoes to rot in storage, the newspaper reported.Growers have been asked to kill all affected plants within 100 feet, and plant samples from both sites will be examined to identify what type of late blight is involved.Experts said that blight can be controlled through the use of fungicides, but the cool, damp weather this summer in the areas has created an ideal environment for the disease to spread.Although Alaska's potato crop is tiny compared to other U.S. states, it has been viewed as a possible global source of seed potatoes in recent years because it is largely free of disease.A local university has explored partnerships with Chinese growers to export certified seed potatoes, noted the Fairbanks Daily News-Miner report.

LOS ANGELES, July 3 (Xinhua) -- Warming of the ocean's subsurface layers will melt underwater portions of the Greenland and Antarctic ice sheets faster than previously thought, increasing the sea level more than already projected, a new study suggests.The subsurface ocean layers surrounding the polar ice sheets will warm substantially as global warming progresses, according to the study led by researchers from the University of Arizona (UA).In addition to being exposed to warming air, underwater portions of the polar ice sheets and glaciers will be bathed in warming seawater, said the study appearing on the website of the American Association for the Advancement of Science (AAAS) on Sunday.The research, based on 19 state-of-the-art climate models, proposes a new mechanism by which global warming will accelerate the melting of the great ice sheets during this century and the next."To my knowledge, this study is the first to quantify and compare future ocean warming around the Greenland and the Antarctic ice sheets using an ensemble of models," said lead author Jianjun Yin, a UA assistant professor of geosciences.According to the study, the subsurface ocean along the Greenland coast could increase as much as 3.6 F (2 C) by 2100.Most previous research has focused on how increases in atmospheric temperatures would affect the ice sheets, he said."Ocean warming is very important compared to atmospheric warming because water has a much larger heat capacity than air," Yin said. "If you put an ice cube in a warm room, it will melt in several hours. But if you put an ice cube in a cup of warm water, it will disappear in just minutes."Given a mid-level increase in greenhouse gases, the researchers found the ocean layer about 650 to 1,650 feet (200 to 500 meters) below the surface would warm, on average, about 1.8 F (1 C) by 2100. Along the Greenland coast, that layer would warm twice as much, but along Antarctica would warm less, only 0.9 F (0.5 C)."No one has noticed this discrepancy before -- that the subsurface oceans surrounding Greenland and Antarctica warm very differently," Yin said.Part of the warming in the North comes from the Gulf Stream carrying warm subtropical waters north. By contrast, the Antarctic Circumpolar Current blocks some of the subtropical warmth from entering the Antarctic's coastal waters.Even so, the Antarctic ice sheet will be bathed in warming waters, according to the study.This paper adds to the evidence that sea level would rise by the end of this century by around one meter and a good deal more in succeeding centuries, the study noted.The study, "Different Magnitudes of Projected Subsurface Ocean Warming Around Greenland and Antarctica," is scheduled for the upcoming edition of Nature Geoscience later this month.

SEOUL, Sept. 26 (Xinhua) -- Samsung Electronics, the world's second-largest manufacturer of mobile phones, launched its first smartphones based on fourth-generation (4G) communication technology in a bid to meet growing demand for high-speed wireless services. Galaxy S2 LTE and Galaxy S2 HD LTE compatible with long- term evolution (LTE) technology were rolled out at a media event held in central Seoul on Monday. The two new smartphones support LTE with data transmission five times faster than the existing third-generation (3G) mobile phones, featuring functions offered by Galaxy S2 smartphones, the company said.The Galaxy S2 LTE is equipped with an Android 2.3, or the latest version of the Android platform, a 4.5-inch wide Super AMOLED display and a 1.5 gigahertz dual core processor, while the Galaxy S2 HD LTE is featuring a 4.65-inch high-definition (HD) AMOLED display with 110 percent natural color reproduction and 180- degree viewing angle. "The 4G LTE technology became the base for enjoying high-speed and high-resolution wireless services. The new products will meet rising demand for such services in an environment where global wireless operators are transitioning to 4G networks," Shin Jong-kyun, president and head of Samsung's mobile communications business, told reporters.Shin forecast sales of the new LTE smartphones would approach the ones of the existing Galaxy S series, adding that it may take time to reach the goal as the LTE networks have yet to be covered nationwide.Global sales of the Galaxy S2 smartphones reached more than 10 million units since its debut in April, according Samsung. The nation's top wireless carrier SK Telecom plans to offer LTE service nationwide by 2013, with the country's No. 3 mobile operator LG Uplus aiming to cover the service across the country next year.

LOS ANGELES, July 18 (Xinhua) -- U.S. scientists have proven that oncogenes can change normal cells into stem-like cells, paving the way to a safer and more practical approach to treating diseases like multiple sclerosis and cancer with stem cell therapy.In a collaborative study, researchers from the Keck School of Medicine of the University of Southern California (USC), and the Children's Hospital of Orange County (CHOC) in California and Good Samaritan Hospital Medical Center in New York have successfully converted human skin cells into brain cells by suppressing the expression of p53, a protein encoded by a widely studied oncogene. This suggests that p53 mutation helps determine cell fate -- good or bad -- rather than only the outcome of cancer.Oncogenes are generally thought to be genes that, when mutated, change healthy cells into cancerous tumor cells.Study findings were appearing Monday on the website of the American Association for the Advancement of Science (AAAS)."The reality may be more complicated than people think," said Jiang F. Zhong, Ph.D., assistant professor of pathology at the Keck School. "What is a stem cell gene? What is a cancer gene? It may be the same thing.""When you turn off p53, people think the cell becomes cancerous because we tend to focus on the bad thing," Zhong said. "Actually, the cell becomes more plastic and could do good things, too. Let's say the cell is like a person who loses his job (the restriction of p53). He could become a criminal or he could find another job and have a positive effect on society. What pushes him one way or the other, we don't know because the environment is very complicated."Stem cells can divide and differentiate into different types of cells in the body. In humans, embryonic stem cells differentiate into three families, or germ layers, of cells. The reasons why and how certain stem cells differentiate into particular layers are not clearly understood. However, from those layers, tissues and organs develop. The endoderm, for example, leads to formation of the stomach, colon and lungs, while the mesoderm forms blood, bone and heart tissue. In its study, Zhong's team examined human skin cells, which are related to brain and neural cells from the ectoderm.When p53 was suppressed, the skin cells developed into cells that looked exactly like human embryonic stem cells. But, unlike other man-made stem cells that are "pluripotent" and can become any other cells in the body, these cells differentiated only into cells from the same germ layer, ectoderm."IPSCs (induced pluripotent stem cells) can turn into anything, so they are hard to control," Zhong said. "Our cells are staying within the ectoderm lineage."Zhong said he expects that suppressing other oncogenes in other families of cells would have the same effect, which could have critical significance for stem cell therapy. Future research should focus on determining which genes to manipulate, Zhong said.The study is slated to appear in the Proceedings of the National Academy of Sciences later this month, according to AAAS.