济南有男科医院(济南检查前列腺的医院) (今日更新中)

BRUSSELS, May 31 (Xinhua) -- A deadly outbreak of E.coli linked to contaminated cucumbers is sending Europe on high alert, as death toll from the disease climbed to 16 till Tuesday.So far, 15 people have reportedly died and more than 300 were seriously ill from infections linked to tainted vegetables in Germany. Experts said the numbers are expected to increase in the coming week."We hope the number of cases will go down but we fear it will worsen," said Oliver Grieve, spokesperson with the University Medical Center Schleswig where most German victims are being treated.The German government has identified the pathogen as hemolytic-uremic syndrome (HUS), a serious complication of a type of E. coli known as Shiga toxin-producing E.coli (STEC). The bacteria could damage blood cells and kidneys, and hence cause deaths.A woman in her 50s was confirmed to be the first fatal victim out of Germany by a local hospital on Tuesday. Officials at the hospital in Boras, southwestern Sweden, said the woman, who had recently traveled to Germany, was admitted on May 29.Swedish health authorities already said on Sunday that 36 people, all linked to travel in northern Germany, had been confirmed to have infected with E.coli, whereas cases were also reported in France, the Netherlands, Denmark and Britain.It was the second food scare for European consumers this year. In January, highly toxic dioxin was found in eggs, poultry and pork products across Germany.

BEIJING, June 14 (Xinhuanet) -- Having a pet at home during the first year of a child may halve the risk of developing allergic to them later in life, a new study suggests.The study was published in the journal Clinical & Experimental Allergy.Researchers from Henry Ford Hospital in Detroit assessed 566 boys and girls who were followed from birth until age 18. They found that boys who had dogs and teens who had cats during their first year of life had 50 percent less risk of developing pet allergies later.Lead researcher Ganesha Wegienka said: "This research provides further evidence that experiences in the first year of life are associated with health status later in life, and that early life pet exposure does not put most children at risk of being sensitised to these animals later in life."The researchers said that exposure to animals at other times in childhood didn't appear to be as significant as the first year.However, Wegienka cautioned that this study doesn't prove a cause-and-effect relationship between having a pet and avoiding allergies, just an association between those two factors."We don't want to say that everyone should go out and get a dog or cat to prevent allergies," she said."More research is needed, though we think this is a worthwhile avenue to pursue."

WASHINGTON, July 20 (Xinhua) -- The loss of a protein that coats sperm may explain a significant proportion of infertility in men worldwide, according to a study by an international team of researchers led by University of California Davis.A paper describing the work was published Wednesday in the journal Science Translational Medicine. The research could open up new ways to screen and treat couples for infertility.The gene DEFB126 encodes a protein called Beta Defensin 126, which coats the surface of sperm and helps it penetrate cervical mucus in the female. A survey of samples from the U.S., Britain and China showed that as many as a quarter of men worldwide carry two copies of the defective gene.In the new study, researchers found that men with a muted DEFB126 lack Beta Defensin 126, making it much more difficult for sperm to swim through the mucus and eventually join with an egg.Examining 500 newly married Chinese couples, researchers found that the lack of Beta Defensin 126 in men with the DEFB126 mutation lowered fertility (even among men that did not display other deficiencies usually associated with infertility, like inadequate semen volume and low sperm motility). Wives of men with the Beta Defensin 126 variant were significantly less likely to become pregnant than were other couples, and 30 percent less likely to have a birth.This genetic variation in DEFB126 likely accounts for many unexplained cases of infertility, researchers say. They hope next to work with a major infertility program in the U.S. to further explore the role of the mutation.

CAIRO, July 6 (Xinhua) -- Egypt's Central Administration of Agricultural Quarantine has denied again that the country's fenugreek seeds exported to Europe caused the E.coli infections which have claimed dozens of lives.The Egyptian authorities took and analyzed fenugreek seeds samples from the warehouse of the said Egyptian exporter and all results were negative by the strain, a statement posted on the official website of Egypt's Agricultural Ministry said on Wednesday."E.coli strain has not been reported in Egypt and no illness cases have been found," it added.If fenugreek seeds sprouts are suspected to be contaminated with E.coli pathogenic strain, it could be related to different handling processes, such as the re-packing or water used for sprouting, the statement said.The European Union (EU) on Tuesday agreed to ban the import of certain types of seeds from Egypt as Egyptian fenugreek seeds were reported to be linked with the outbreak of E.coli in Europe.The European Food Safety Authority (EFSA) published early Tuesday a report from its task force on the E.coli outbreaks in Germany and France, indicating that fenugreek seeds imported from Egypt is the most likely source of the outbreaks.The E.coli outbreak has killed over 50 people in Europe since it was first reported in late May.Russia's food safety watchdog Rospotrebnadzor has also banned the import of certain types of produce from Egypt over food safety concerns, a Russian official said on Wednesday.Egypt's Ministry of Agriculture refuted the claims of Egypt being the source of E.coli Friday.

WELLINGTON, June 7 (Xinhua) -- Scientists in New Zealand have developed a new drug to fight previously untreatable hypoxic cancer tumors, which form in areas of the body starved of oxygen.The researchers at the Auckland University have entered an agreement for the clinical development of CEN-209, which was developed over 10 years of research, said a statement from the university Tuesday.CEN-209 was designed to enhance the effectiveness of radiotherapy and chemotherapy in solid hypoxic tumors, which were resistant to standard cancer therapies, said the statement. In lung cancer patients for example, about half of tumors had hypoxic regions.The new drug worked by damaging the DNA of hypoxic cancer cells, while leaving normal, healthy tissues alone.CEN-209 was designed and created by researchers at the university's Auckland Cancer Society Research Centre (ACSRC), using computer models of drug transport within tumors to accurately predict the anti-tumor activity of the drugs."Our computer models of drug transport developed in-house allowed the synthetic chemists to test their design theories and considerably shortened the discovery process," said Associate Professor Michael Hay, who led the ACSRC research chemists."CEN-209 improves markedly on previous agents in this class in terms of its ability to penetrate tumors, and this is reflected by its improved activity in the laboratory, when combined with long or short courses of radiotherapy," said researcher Professor Bill Wilson.Under the agreement between the university's Auckland UniServices Ltd. and California-based Centella Therapeutics, Inc., a subsidiary of Varian Medical Systems, Inc., Centella will have exclusive rights to CEN-209, which it will develop and trial with Cancer Research UK.The work on CEN-209 is the culmination of a program initiated with funding from the U.S. National Cancer Institute, and more recently from the Maurice Wilkins Centre for Biodiscovery. Ongoing preclinical research on CEN-209 and a backup compound was funded by grants from the Auckland Medical Research Foundation, Genesis Oncology Trust and Health Research Council of New Zealand, said the statement.

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