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济南早泄有的治疗吗
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发布时间: 2025-05-31 06:55:47北京青年报社官方账号
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  济南早泄有的治疗吗   

CANBERRA, Aug. 29 (Xinhua) -- Alzheimer Australia on Monday released a GPS device for people with dementia, in a move to give them greater independence, while reducing the burden of missing person searches for police.The technology has been using in New South Wales of Australia, and is now available for people in state Victoria.The Safe2Walk locater can be worn by people with dementia on a lanyard around the neck or clipped onto a belt. Families can log- on to the connected website and see where the person is.The device updates the person's location every 60 seconds, reducing stress for carers so they know when a person with dementia might be wandering.It also works as a mobile phone, letting the person wearing the device to make instant calls to family.According to Alzheimer's Australia's research manager Jason Burton, the device aimed to stop vulnerable people getting lost, with research showing about 40 percent of people with dementia went missing at least once."In 99 percent of cases the carer has gone to pick them up, but there was one case where they couldn't and the police were able to contact us to get the exact GPS location of this person to rescue them," Burton told Herald Sun.A Victoria Police spokeswoman said while it could not endorse a specific product, if the device could alert carers when a person with dementia first became disorientated, the response could help avoid a large-scale police search.Mina Sapounakis, who's father has worn the Safe2Walk device, said it has given her family a sense of calm."We could go grocery shopping without stressing and rushing back home quickly to check on Dad," she said."There were a few times he had gone wandering and we were able to easily find him without having to call the police."The Safe2Walk GPS costs under 15 U.S. dollars a week for rent.

  济南早泄有的治疗吗   

WASHINGTON, June 6 (Xinhua) -- Women who have higher levels of the appetite-controlling hormone leptin have fewer symptoms of depression, and this apparent inverse relationship is not related to body mass index (BMI), a new study finds. The results were presented Monday at The Endocrine Society's 93rd Annual Meeting in Boston, the United States."Animal data suggest that leptin may reduce anxiety and improve depression. Our study in women suggests that leptin may indeed have antidepressant qualities," said the study's lead author, Elizabeth Lawson, of Massachusetts General Hospital and Harvard Medical School in Boston.Leptin, the product of fat cells, signals satiety, or fullness. It is low in thin women and high in obese women, according to Lawson. She also said there is an increased prevalence of anxiety and depression in certain conditions in which leptin levels are typically low. These include the eating disorder anorexia nervosa, in which there is abnormally low weight and body fat, and functional hypothalamic amenorrhea, in which women have stopped menstruating despite having normal weight."It is unknown whether low leptin levels contribute to the development of mood disorders in these women," Lawson said.She and her co-workers studied the relationship between leptin levels and symptoms of anxiety and depression in 64 women. Fifteen of the women had anorexia nervosa, 12 were normal weight with hypothalamic amenorrhea, 20 were normal weight and in good health, and 17 were overweight or obese but still healthy.All subjects were asked questions to assess symptoms of depression and anxiety, with high scores indicating more symptoms. Besides measuring leptin levels in the blood, the researchers assessed the women's BMI, a measure of weight for height.The relationship between leptin and depression symptoms was independent of BMI. This finding indicates that leptin may mediate symptoms of depression and that this effect is not a function of low weight, Lawson said."Further research administering leptin to humans will be important in understanding whether this hormone has a potential role in the treatment of depression," she said.

  济南早泄有的治疗吗   

  

CANBERRA, Sept. 7 (Xinhua) -- A team of Australian and U.S. scientists on Wednesday said they discovered a genetic defect, which can lead to Leigh syndrome, a rare disorder which affects the central nervous syndrome.The scientists tested more than 1000 genes by encoding proteins active in two individuals who suffer from the illness. They used a new technique known as next-generation DNA sequencing to examine the genes.The gene they discovered encodes an enzyme which is found in the mitochondria which are subsets of cells. Without this enzyme the mitochondria do not translate proteins efficiently, and this then causes Leigh syndrome.According to David Thorburn, from the Murdoch Children's Research Institute in Melbourne, the discovery shows the enormous potential of the new technology."These findings demonstrate the ability of sequencing technologies to improve diagnosis," Professor Thorburn said in a statement released on Wednesday."Although it isn't clear in the case of Leigh syndrome whether the precise molecular diagnosis will necessarily lead to therapies, the current findings represent a meaningful service."He added that diagnosis of the disease along with its specific genetic cause can also be informative about the risk a couple has of having another affected child. The diagnostic information can help in decisions about whether and how to pursue alternative means of having children, for instance through the use of donor sperm or eggs.The research team consisted of scientists from Australia's Murdoch Institute as well as the Broad Institute in the U.S.In Leigh syndrome, infants are born apparently healthy only to develop movement and breathing disorders that worsen over time, often leading to death by the age of three. The problem is that the mitochondria responsible for powering their cells cannot keep up with the demand for energy in their developing brains.

  

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