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WASHINGTON, Sept. 13 (Xinhua) -- The U.S. Department of Health and Human Services (HHS), with several partners, on Tuesday launched Million Hearts, an initiative that aims to prevent one million heart attacks and strokes over the next five years.The program will focus on helping Americans make healthy choices, such as preventing tobacco use and lowering consumption of salt and trans fats, and increasing use of treatments like aspirin and blood pressure and cholesterol-lowering medications.The HHS hopes that by 2017, 65 percent of high-risk patients will be taking aspirin and have their blood pressure and cholesterol under control. Currently, only 47 percent of high-risk patients take aspirin, and only 33 percent have their cholesterol and 46 percent their blood pressure under control.They also aim to cut smoking to 17 percent of Americans from 19 percent by 2017, and seek a 20 percent drop in sodium intake and a 50 percent drop in trans fat consumption."Heart disease causes one of every three American deaths and constitutes 17-percent of overall national health spending," said HHS Secretary Kathleen Sebelius in a statement. "By enlisting partners from across the health sector, Million Hearts will create a national focus on combating heart disease."

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."

BEIJING, Aug. 5 (Xinhuanet) -- A study conducted by Israeli researchers at the Albert Einstein College of Medicine in New York shows that genes, rather than good eating and lifestyle habits, determine longevity, according to media reports Friday. "This study suggests that centenarians may possess additional longevity genes that help to buffer them against the harmful effects of an unhealthy lifestyle," said senior author Nir Barzilai, director of the Institute for Aging Research at Yeshiva University's Albert Einstein College of Medicine.The study involves 477 Ashkenazi Jews between the ages of 95 and 122, of whom 75 percent are women. According to the study, Ashkenazi Jews are chosen as subjects because they are more "genetically uniform than other populations, making it easier to spot gene differences that are present."The study also found that the long-lived Ashkenazi Jews drank slightly more and exercised less than their average counterparts. But Barzilai also warned: “Although this study demonstrates that centenarians can be obese, smoke and avoid exercise, those lifestyle habits are not good choices for most of us who do not have a family history of longevity.""We should watch our weight, avoid smoking and be sure to exercise, since these activities have been shown to have great health benefits for the general population, including a longer lifespan," He added.

BEIJING, Sept. 21 (Xinhuanet) -- China will launch an unmanned module next week, paving the way for a planned space station, a spokesman for the space program said on Tuesday.Tiangong-1, or "Heavenly Palace 1", will blast off from Jiuquan Satellite Launch Center in Gansu province between Sept 27 and 30, the spokesman said.The 8.5-ton module, and the Long March II-F rocket that will carry it skyward, were positioned onto the launch pad on Tuesday, signaling that the project has entered the final preparation stage."Scientists will conduct final tests in the next few days before injecting propellants for the launch," Cui Jijun, director of the Jiuquan Satellite Launch Center, said.The program spokesman said that Tiangong-1 will serve as "a target spacecraft" for rendezvous and docking experiments.It will also work as "a platform to test long-term unmanned and short-term manned operations", he said.Technical and medical experiments will provide crucial data ahead of the building of the space station (scheduled for 2020).Three rendezvous and docking experiments will be conducted.An unmanned Shenzhou-VIII craft will be launched later this year to dock with Tiangong-1. In 2012, Shenzhou IX and Shenzhou X will blast off to complete at least one manned docking.Jiao Weixin, a space scientist at Peking University, said that rendezvous and docking - vital maneuvers for manned programs - are hard to master."Rendezvous and docking are difficult because it is like asking two racing cars to keep a distance of 1 meter between them," he said.The mission requires two craft, traveling at speeds of 28,000 km/h, to enter the same orbit and connect with precision, he said.The China Manned Space Engineering Office rescheduled the launch of Tiangong-1 after the failed launch of an experimental orbiter last month.The spokesman said that the carrier rocket to blast off with Tiangong-1 atop has been modified.Only the United States and Russia have mastered rendezvous and docking technology, said Pang Zhihao, deputy editor-in-chief of the magazine Space International.Spacecraft developed by Europe and Japan have utilized US and Russian technology to dock with the International Space Station, he added.