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BEIJING, Sept. 1 (Xinhua) -- Chinese scientists have decided to reschedule the launch of the Tiangong-1, an unmanned space module, due to the failed launch of an experimental orbiter, a spokesperson with the project said Thursday.The decision to delay the launch was based on the consideration that the carrier rocket Long-March II-F, which would be used for the upcoming launch, belongs to the same series as the malfunctioning one that led to the experimental orbiter SJ-11-04 failing to enter Earth's orbit in August."As the investigation into the malfunction of the rocket Long-March II-C remains underway, it is not clear yet whether the malfunction is linked with the Long-March II-F," the unnamed spokesperson said.Previously, the Long-March II-F was expected to send the 8.5-metric ton Tiangong-1, or "Heavenly Palace 1," into space in the second half of this year to perform the nation's first space-docking procedure.The Tiangong-1 will dock with the unmanned Shenzhou-8 spacecraft, which will be sent into space after the Tiangong-1's launch."The specific launch date for the Tiangong-1 will be set based on the results of the investigation into the malfunctioning rocket," the spokesperson said, adding that the project's teams are currently double-checking every product that will be involved in the space-docking.The Shenzhou-8 spacecraft, the Long March II-F carrier rocket, and the Tiangong-1 have all been transferred to the Jiuquan Satellite Launch Center in northwest China's Gansu Province, the spokesperson said.

WASHINGTON, Aug. 3 (Xinhua) -- The annual number of new HIV infections in the United States was relatively stable at approximately 50,000 new infections each year between 2006 and 2009, the Centers for Disease Control and Prevention's first multi- year estimates from its national HIV incidence surveillance find.However, HIV infections increased among young men who have sex with men (MSM) between 2006 and 2009, driven by alarming increases among young, black MSM -- the only subpopulation to experience a sustained increase during the period.According to the new estimates, there were 48,600 new HIV infections in the United States in 2006, 56,000 in 2007, 47,800 in 2008 and 48,100 in 2009. The multi-year incidence estimates allow for a reliable examination of trends over time. They reveal no statistically significant change in HIV incidence overall from 2006 to 2009, with an average of 50,000 for the four-year period. In 2009, the largest number of new infections was among white MSM (11,400), followed closely by black MSM (10,800). Hispanic MSM (6, 000) and black women (5,400) were also heavily affected.The new estimates were published Wednesday in the on-line scientific journal PLoS ONE."More than 30 years into the HIV epidemic, about 50,000 people in this country still become infected each year. Not only do men who have sex with men continue to account for most new infections, young gay and bisexual men are the only group in which infections are increasing, and this increase is particularly concerning among young African American MSM," said CDC Director Thomas Frieden. " HIV infections can be prevented. By getting tested, reducing risky behaviors, and getting treatment, people can protect themselves and their loved ones."

CANBERRA, Aug. 31 (Xinhua) -- Australia's new chief medical officer, Professor Chris Baggoley, on Wednesday said Australian should not be worried about reports that a mutant strain of the deadly bird flu virus is spreading across Asia and beyond.On Monday, the UN's Food and Agriculture Organization (FAO) said a mutant strain of the deadly avian influenza virus was spreading in Asia and issued a warning that the bird flu could spread from poultry to humans. It urged "heightened readiness and surveillance" as the mutant strain posed "unpredictable risks to human health."But Professor Baggoley, who took office on Tuesday, said Australia is well served by strict testing and customs regimes, adding that avian migration patterns also mean infected birds are highly unlikely to arrive in Australia."I don't think we should be worried," Prof Baggoley told Sky News."The country and the Australian government is certainly vigilant in relation to bird flu."Prof. Baggoley insisted the H5N1 virus remains overwhelmingly a disease of birds and it is very uncommon for humans to catch it. It is also exceedingly rare for humans to spread it among themselves.He said that even if bird flu did arrive and affected humans, Australia is ready to respond to any new and significant developments in bird flu."Australia tests migratory wild birds, looking for H5N1, and has been doing this for some years and has never found it," he said."On the human side of things, Australia has been preparing for a possible outbreak of H5N1 since 2004."We've got the laboratory capacity to diagnose influenza; we've got arrangements in place for a rapid production of an H5N1 vaccine for humans, and we've got ready access to antivirals."There have been no outbreaks of bird flu in Australia to date.The UN said the latest human death from bird flu occurred earlier this month in Cambodia, which has registered eight cases of human infection this year, all of them fatal.H5N1 has infected 565 people since it first appeared in 2003, killing 331.

WASHINGTON, Sept. 12 (Xinhua) -- An enzyme that appears to play a role in controlling the brain's response to nicotine and alcohol in mice might be a promising target for a drug that simultaneously would treat nicotine addiction and alcohol abuse in people, U.S. researchers find.Over the course of four weeks, mice genetically engineered to lack the gene for protein kinase C (PKC) epsilon consumed less of a nicotine-containing water solution than normal mice, and were less likely to return to a chamber in which they had been given nicotine. In contrast, normal mice steadily increased their consumption of nicotine solution while the mice lacking PKC epsilon did not.The study conducted by researchers at the Ernest Gallo Clinic and Research Center, affiliated with the University of California, San Francisco, appeared Monday in the online edition of the Proceedings of the National Academy of Sciences.In normal mice, as in humans, nicotine binds to a certain class of nicotinic receptors located on dopamine neurons, which causes dopamine to be released in the brain. Dopamine creates a feeling of enjoyment, and thus prompts a sense of reward. Researchers found that mice lacking PKC epsilon are deficient in these nicotinic receptors.The finding complements earlier research in which researchers found that mice genetically engineered to lack the PKC epsilon enzyme drank less alcohol than normal mice and were disinclined to return to a chamber in which they had been given alcohol."This could mean that these mice might not get the same sense of reward from nicotine or alcohol," said Gallo senior associate director and investigator Robert Messing. "The enzyme looks like it regulates the part of the reward system that involves these nicotinic receptors."The reward system is a complex of areas in the brain that affect craving for nicotine, alcohol and other addictive substances.The next step in the research, said Messing, would be to develop compounds that inhibit PKC epsilon. The ultimate goal, he added, would be medications that could be used "to take the edge off of addiction by helping people get over some of their reward craving."