呼和浩特那家肛裂手术做得好(呼和浩特结肠息肉怎么治疗) (今日更新中)

VIENNA, Sept. 9 (Xinhua) -- The three-day Traditional Chinese Medicine (TCM) exhibition opened Friday in the Flower Garden Hirschstetten in Vienna, with the aim to introduce traditional Chinese medicine to the Austrians and to arouse their interest in the culture of Chinese medicine.Holding for the second time, the exhibition this year have attracted more visitors to take part in the health talks on traditional Chinese medicine, read or buy books around the theme of traditional Chinese medicine. Some visitors enthusiastically tried Chinese diet therapy prepared by the organizers.In recent years, traditional Chinese medicine has been increasingly recognized and accepted in Austria. It has already not only entered into the classroom but also into the hospital and clinic.Some private health insurance companies in Austria have started to include the treatment by traditional Chinese medicine into their insuring categories. Chinese patent drugs are already available in many pharmacies in Austria.But in general, traditional Chinese medicine still faces many limits in Austria, which hasn't been involved into the public health insurance system. Some Austrians still have doubts of Chinese medicine, in particular the medicinal herb drugs. All these have restricted the development of traditional Chinese medicine here.Currently, major clinics of traditional Chinese medicine carry out only acupuncture and moxibustion, massage and other health-care physical therapy.Richard Schmerker, a doctor of traditional Chinese medicine in Austria, told reporters that the development of traditional Chinese medicine here still faces many challenges and the biggest one is the shortage of effective propaganda and popularization.But he expressed his full confidence about the future of traditional Chinese medicine in Austria. He said, the uniqueness of finding the root cause of diseases and laying the axe to the root will make it be accepted by more and more Austrians.

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."

WASHINGTON, July 13 (Xinhua) -- Human neural stem cells are capable of helping people regain learning and memory abilities lost due to radiation treatment for brain tumors, a University of California, Irvine (UCI) study suggests.Research with rats found that stem cells transplanted two days after cranial irradiation restored cognitive function, as measured in one- and four-month assessments. In contrast, irradiated rats not treated with stem cells showed no cognitive improvement."Our findings provide solid evidence that such cells can be used to reverse radiation-induced damage of healthy tissue in the brain," said Charles Limoli, a UCI radiation oncology professor.Study results will appear Friday in Cancer Research, a journal of the American Association for Cancer Research.Radiotherapy for brain tumors is limited by how well the surrounding tissue tolerates it. Patients receiving radiation at effective levels suffer varying degrees of learning and memory loss that can adversely affect their quality of life."In almost every instance, people experience severe cognitive impairment that's progressive and debilitating," Limoli said. " Pediatric cancer patients can experience a drop of up to three IQ points per year."For the UCI study, multipotent human neural stem cells were transplanted into the brains of rats that had undergone radiation treatment. They migrated throughout the hippocampus -- a region known for the growth of new neurons -- and developed into brain cells.Researchers assessed the rats one month and four months after transplantation, noting enhanced learning and memory abilities at both intervals.Additionally, they found that transplanting as few as 100,000 human neural stem cells was sufficient to improve cognition after cranial irradiation. Of cells surviving the process, about 15 percent turned into new neurons, while another 45 percent became astrocytes and oligodendrocytes -- cells that support cerebral neurons.Most notably, Limoli said, he and his colleagues discovered that about 11 percent of the engrafted cells expressed a behaviorally induced marker of learning, indicating the functional integration of those cells into memory circuits in the hippocampus."This research suggests that stem cell therapies may one day be implemented in the clinic to provide relief to patients suffering from cognitive impairments incurred as a result of their cancer treatments," Limoli said.

WASHINGTON, June 16 (Xinhua) -- Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage. New research by a team of scientists at the University of Rochester has unveiled an important new mechanism that allows cells to recognize when they are under stress and prime the DNA repair machinery to respond to the threat of damage.Their findings will be published Friday in journal Science. Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage.The scientists, led by biologists Vera Gorbunova and Andrei Seluanov, focused on the most dangerous type of DNA damage -- double strand breaks. Unrepaired, this type of damage can lead to premature aging and cancer. They studied how oxidative stress affects efficiency of DNA repair. Oxidative stress occurs when the body is unable to neutralize the highly-reactive molecules, which are typically produced during routine cellular activities.The research team found that human cells undergoing oxidative stress synthesized more of a protein called SIRT6. By increasing SIRT6 levels, cells were able to stimulate their ability to repair double strand breaks. When the cells were treated with a drug that inactivated SIRT6, DNA repair came to a halt, thus confirming the role of SIRT6 in DNA repair. Gorbunova notes that the SIRT6 protein is structurally related to another protein, SIR2, which has been shown to extend lifespan in multiple model organisms."SIRT6 also affects DNA repair when there is no oxidative stress," explains Gorbunova. "It's just that the effect is magnified when the cells are challenged with even small amounts of oxidative stress."SIRT6 allows the cells to be economical with their resources, priming the repair enzymes only when there is damage that needs to be repaired. Thus SIRT6 may be a master regulator that coordinates stress and DNA repair activities, according to Gorbunova.

XICHANG, Sichuan, Aug. 12 (Xinhua) -- China launched a communications satellite PAKSAT-1R for Pakistan at 0:15 a.m. Friday from the Xichang Satellite Launch Center in southwest China's Sichuan Province.The satellite was carried by a Long March-3B carrier rocket, according to the launch center. It is China's first in-orbit delivery to Asian customers and also the first commercial satellite export to international users this year.According to statistics from the control center, the satellite successfully separated from its carrier rocket and entered geostationary transfer orbit as scheduled, 26 minutes after being launched.PAKSAT-1R will provide a range of services, including broadband Internet, telecom and broadcasting, covering some regions of Europe, South Asia, the Middle East, and the eastern Africa.The contract for the PAKSAT-1R was signed in 2008 between China Great Wall Industry Corporation and the Space and Upper Atmosphere Research Commission of Pakistan.China and Pakistan share a long history of space technology cooperation. Pakistan's first low-orbit satellite, BADR-A, was launched by China in 1990 with Long March 2E rocket.

来源：资阳报