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CANBERRA, Sept. 7 (Xinhua) -- A team of Australian and U.S. scientists on Wednesday said they discovered a genetic defect, which can lead to Leigh syndrome, a rare disorder which affects the central nervous syndrome.The scientists tested more than 1000 genes by encoding proteins active in two individuals who suffer from the illness. They used a new technique known as next-generation DNA sequencing to examine the genes.The gene they discovered encodes an enzyme which is found in the mitochondria which are subsets of cells. Without this enzyme the mitochondria do not translate proteins efficiently, and this then causes Leigh syndrome.According to David Thorburn, from the Murdoch Children's Research Institute in Melbourne, the discovery shows the enormous potential of the new technology."These findings demonstrate the ability of sequencing technologies to improve diagnosis," Professor Thorburn said in a statement released on Wednesday."Although it isn't clear in the case of Leigh syndrome whether the precise molecular diagnosis will necessarily lead to therapies, the current findings represent a meaningful service."He added that diagnosis of the disease along with its specific genetic cause can also be informative about the risk a couple has of having another affected child. The diagnostic information can help in decisions about whether and how to pursue alternative means of having children, for instance through the use of donor sperm or eggs.The research team consisted of scientists from Australia's Murdoch Institute as well as the Broad Institute in the U.S.In Leigh syndrome, infants are born apparently healthy only to develop movement and breathing disorders that worsen over time, often leading to death by the age of three. The problem is that the mitochondria responsible for powering their cells cannot keep up with the demand for energy in their developing brains.

SAN FRANCISCO, June 13 (Xinhua) -- Social networking giant Facebook will likely go public in the first quarter of 2012 with a valuation that could top 100 billion U.S. dollars, U.S. media reported on Monday.In a report, CNBC quoted people familiar with the matter as saying that Facebook could submit filing to register its securities with the U.S. Securities and Exchange Commission (SEC) as early as October or November this year.People who are on Wall Street and track this information told the business news television channel that they think the Facebook initial public offering (IPO), if and when it happens could value the company of more than 100 billion dollars.A factor in the company's IPO timing is the SEC's requirement that companies must disclose financial information if they have more than 500 private investors."The company has until the end of April 2012 to disclose their financials, but they may just want to get ahead of that by doing a formal initial public offering, I'm told. And that could happen in the first quarter of the year," said CNBC Wall Street reporter Kate Kelly.Facebook is also facing internal pressure as employees have not been permitted to sell their private shares on the secondary market since last spring. An IPO would make it easier for employees to monetize their shares, said Kelly, citing sources.Facebook shares have been traded in private markets such as Sharepost.com, which puts the social networking company's valuation at 85 billion dollars.Meanwhile, latest data show that Facebook is losing users last month in the United States, Canada and several European countries, indicating that the company could have hit the limits of expansion in its mature markets.

LOS ANGELES, July 18 (Xinhua) -- U.S. scientists have proven that oncogenes can change normal cells into stem-like cells, paving the way to a safer and more practical approach to treating diseases like multiple sclerosis and cancer with stem cell therapy.In a collaborative study, researchers from the Keck School of Medicine of the University of Southern California (USC), and the Children's Hospital of Orange County (CHOC) in California and Good Samaritan Hospital Medical Center in New York have successfully converted human skin cells into brain cells by suppressing the expression of p53, a protein encoded by a widely studied oncogene. This suggests that p53 mutation helps determine cell fate -- good or bad -- rather than only the outcome of cancer.Oncogenes are generally thought to be genes that, when mutated, change healthy cells into cancerous tumor cells.Study findings were appearing Monday on the website of the American Association for the Advancement of Science (AAAS)."The reality may be more complicated than people think," said Jiang F. Zhong, Ph.D., assistant professor of pathology at the Keck School. "What is a stem cell gene? What is a cancer gene? It may be the same thing.""When you turn off p53, people think the cell becomes cancerous because we tend to focus on the bad thing," Zhong said. "Actually, the cell becomes more plastic and could do good things, too. Let's say the cell is like a person who loses his job (the restriction of p53). He could become a criminal or he could find another job and have a positive effect on society. What pushes him one way or the other, we don't know because the environment is very complicated."Stem cells can divide and differentiate into different types of cells in the body. In humans, embryonic stem cells differentiate into three families, or germ layers, of cells. The reasons why and how certain stem cells differentiate into particular layers are not clearly understood. However, from those layers, tissues and organs develop. The endoderm, for example, leads to formation of the stomach, colon and lungs, while the mesoderm forms blood, bone and heart tissue. In its study, Zhong's team examined human skin cells, which are related to brain and neural cells from the ectoderm.When p53 was suppressed, the skin cells developed into cells that looked exactly like human embryonic stem cells. But, unlike other man-made stem cells that are "pluripotent" and can become any other cells in the body, these cells differentiated only into cells from the same germ layer, ectoderm."IPSCs (induced pluripotent stem cells) can turn into anything, so they are hard to control," Zhong said. "Our cells are staying within the ectoderm lineage."Zhong said he expects that suppressing other oncogenes in other families of cells would have the same effect, which could have critical significance for stem cell therapy. Future research should focus on determining which genes to manipulate, Zhong said.The study is slated to appear in the Proceedings of the National Academy of Sciences later this month, according to AAAS.

BERLIN, Aug. 28 (Xinhua) -- Mercedes-Benz, the world leading luxury car producer, celebrates the 125th anniversary of its invention of automobile with what it claimed the largest gathering ever, at the former Tempelhof airport in Berlin on the weekend.The Mercedes-Benz Club and Mercedes-Benz enthusiasts greet visitors with some 1,800 well-assorted Mercedes-Benz-branded vehicles, ranging from vintage and classic passenger cars to a variety of historic commercial vehicles, on a vast open areas of 250,000 square metres of the abandoned Tempelhof airport, which was closed in 2008.It took Mr. Lauritzen and his wife two days' drive of their cherised wecker of Mercedes-Benz Cabriolet B. 320.W 142.-3,4l to reach here for taking part in the pageant."It is a long journey, but we are happy to be here to see more class cars and more friends with same interests," Mrs Lauritzen said.Owners take the opportunity to present their lovingly cared-for vehicles, which include a number of truly rare items of automotive history, to the Berlin public.The couple of Mr. Kerner drove their red Mercedes-Benz 170Dba, a made-in-1952 model with a range of special and unique designs which are very rarely seen now, over journey of 155 km from Bitterfeld to join in the gala."I'm fond of driving my classic car of Benz," said Mrs Kerner, despite it can run maximally at a speed of 50 to 80 km per hour, "but I truly enjoy the diesel engine sounds and the historic feeling it evokes."The legendary Silver Arrows, dating from the 1930s and 1950s, are also presented from the Mercedes-Benz Classic collection for demonstration drives."The brilliant history of the Silver Arrows will be brought to life with a demonstration of their impressive engine sound and incredible driving dynamics," said Michael Bock, head of Mercedes-Benz Classic and Director of Mercedes-Benz Museum GmbH."It is not often that these racing cars are driven in public, so this will be a true highlight of the program of events in Tempelhof."During the grand show, visitors are also accessible to first-hand experiences of the latest technologies applicable in the future for in passenger cars, commercial vehicles and racing cars."Some 125 years ago, Carl Benz submitted his patent application for the Benz Patent Motor Car to the Patent Office in Berlin", the organizer appealed to the visitors, saying now Mercedes-Benz is returning to Berlin to celebrate together with Berliners.