中山便血怎么诊疗(中山怎么治好痔疮) (今日更新中)

LONDON, Sept. 22 (Xinhua) -- A British publisher on Thursday issued an "unauthorized autobiography" of the founder of the controversial Wikileaks website Julian Assange.Assange became a global figure after he published 250,000 secret United States diplomatic cables on his Wikileaks website, which became a serious embarrassment to the American government.He was then accused by two women of rape when he was in Sweden. Swedish police said they wanted to question him, and issued a European Arrest Warrant in 2010 for him.Assange, 40, denies the allegations and surrendered himself to police in London at the end of 2010, and the Swedish authorities applied for his extradition to face questioning.Assange fought the extradition bid in the English courts, fearing that he could face further extradition from Sweden to the United States where he could face criminal charges related to the publishing of the secret cables, but failed.He appealed against the extradition ruling in July and a final decision on the case will be made by senior English judges, probably before Christmas.Assange agreed to cooperate with Edinburgh-based publisher Canongate in publishing an autobiography and had 50 hours of interviews with a ghostwriter between January and March this year, while he was on bail awaiting an outcome of the extradition hearings.He received a 500,000 pound advance (about 768,000 U.S. dollars) for the book.Publisher's spokesman Liz Sich told Xinhua Thursday, "It's an unauthorized autobiography -- it is his words. He was contracted to write his autobiography in December; a ghostwriter was assigned to it, approved by the publisher and Julian and an intense amount of work was done in the first three months of 2011. The first draft was delivered on schedule at the end of March. After that there was an hiatus and nothing happened; in June Julian decided he wanted to tear up the contract."Assange has opposed publication, but Sich said, "It is very much Julian's words, it is written in the first person. He didn't want it to be published but he was in breach of his contract. He couldn't pay the advance back because he had used it to pay his lawyers."The book is available only in English at the moment, but a Dutch publisher and a Turkish publisher said they would print translations in their languages, and other foreign language editions are also likely.

BEIJING, August 4 (Xinhuanet) -- Medtronic Inc. is giving Yale University a 2.5 million U.S. dollar grant to review the safety and effectiveness of its controversial spine treatment called Infuse Bone Graft, according to The Wall Street Journals Thursday.Under terms of the pact, the company will release to Yale and eventually the public what participants described as an unprecedented amount of clinical trial and other data. The university will select two research teams from other organizations to perform separate analyses of the data.The announcement comes in the wake of a paper in June in the Spine Journal. The paper revealed that several clinical studies of Infuse, conducted by surgeons with strong economic ties to Medtronic, failed to report serious complications that arose in the trials. The critical paper also found that many of the studies were designed in ways in favor of Infuse over a treatment given to a control group.Medtronic disputes some of the findings and is conducting an internal investigation of the matter.Infuse accounts for about 700 million dollars in annual sales for Medtronic. The product is under investigation by the Senate Finance Committee over the role payments to physicians who may have played how studies were reported.

SYDNEY, Aug. 25 (Xinhua) -- Scientists in Australia have discovered a way of stopping mosquitoes carrying dengue virus, raising hopes for preventing the 50 million human cases of the disease every year, local media reported on Thursday.Groundbreaking experiments in Queensland have found a common insect bacteria, wMel Wolbachia, which can dramatically reduce the presence of dengue fever in mosquitoes.The research, led by Professor Scott O'Neill, Dean at Melbourne's Monash University, was published on Thursday in the prestigious journal, Nature.Australian researchers working on the Eliminate Dengue program aim to protect the mosquitoes themselves from dengue and so stop them transmitting the virus to humans."What the experiments have shown is that this strain of Wolbachia when it is put into mosquitoes really reduces the ability of the (dengue) virus to grow in the mosquito and if it can't grow, then it can't get transmitted in people," O'Neill told reporters.O'Neill said while it was too early to say if the experiments heralded the end of dengue fever, it was a major step towards that goal.In the past decade, there have been 2400 cases of dengue fever reported during 36 outbreaks in Australia.Dengue fever has become endemic in tropical regions, where it is spread by a specific type of mosquito that becomes infected after biting humans with the disease.Despite millions of people being infected with dengue each year, there is currently no way of stopping its rapid spread either by vaccines or controlling mosquito populations.Further trials will be conducted in Cairns in north Queensland over the coming wet season and approval is currently being sought for trials in Thailand, Vietnam, Brazil and Indonesia that will directly determine the effectiveness of the method in reducing dengue disease in human populations, according to Monash University.

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."

LOS ANGELES, June 7 (Xinhua) -- Salmonella, not E. coli, poses the greatest health threat to Americans, the U.S. Centers for Disease Control and Prevention (CDC) announced on Tuesday.Salmonella infections have risen 10 percent over the past 15 years, while several types of foodborne illness have been falling, the agency said in a report.Infections from Shiga toxin-producing E. coli O157 (the strain of most concern in the United States) have dropped almost in half and the rates of six other foodborne infections have been cut 23 percent, the CDC said.The agency issued the report as a deadly new strain of E.coli is spreading in several European countries."There are about 50 million people each year who become sick from food in the U.S.. That's about one in six Americans," CDC director Dr. Thomas R. Frieden said.The CDC attributed the reduction in infections from E. coli O157 to better detection and investigation of outbreaks, cleaner slaughterhouse methods, better testing of ground beef for E. coli, improved inspections of ground beef processing plants, regulations prohibiting E. coli O157 in ground beef and increased awareness of the importance of properly cooking beef.Other foodborne illnesses that fell in incidence over the same time period include those caused by the campylobacter, listeria, vibrio and yersinia pathogens.To reduce their risk of foodborne illness, people should assume that raw chicken and other meat have bacteria that can make you sick, the CDC said.In the kitchen, raw meats should not allow to contaminate counter tops or cutting boards and should be kept away from other foods, such as fruits and vegetables, the CDC advised.The agency also called on people to thoroughly cook meat, poultry, eggs and shellfish, while refraining from consuming unpasteurized milk, juice or soft cheese.

来源：资阳报