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WASHINGTON, June 14 (Xinhua) -- Eating a low-carbohydrate, high- protein diet may reduce the risk of cancer and slow the growth of tumors already present, according to a study published Tuesday in Cancer Research, a journal of the American Association for Cancer Research.The study was conducted in mice, but the scientists involved agree that the strong biological findings are definitive enough that an effect in humans can be considered."This shows that something as simple as a change in diet can have an impact on cancer risk," said lead researcher Gerald Krystal, a scientist at the British Columbia Cancer Research Center.Krystal and his colleagues implanted various strains of mice with human tumor cells or with mouse tumor cells and assigned them to one of two diets. The first diet, a typical Western diet, contained about 55 percent carbohydrate, 23 percent protein and 22 percent fat. The second, which is somewhat like a South Beach diet but higher in protein, contained 15 percent carbohydrate, 58 percent protein and 26 percent fat. They found that the tumor cells grew consistently slower on the second diet.As well, mice genetically predisposed to breast cancer were put on these two diets and almost half of them on the Western diet developed breast cancer within their first year of life while none on the low-carbohydrate, high-protein diet did. Interestingly, only one on the Western diet reached a normal life span ( approximately 2 years), with 70 percent of them dying from cancer while only 30 percent of those on the low-carbohydrate diet developed cancer and more than half these mice reached or exceeded their normal life span.Krystal and colleagues also tested the effect of an mTOR inhibitor, which inhibits cell growth, and a COX-2 inhibitor, which reduces inflammation, on tumor development, and found these agents had an additive effect in the mice fed the low-carbohydrate, high-protein diet.When asked to speculate on the biological mechanism, Krystal said that tumor cells, unlike normal cells, need significantly more glucose to grow and thrive. Restricting carbohydrate intake can significantly limit blood glucose and insulin, a hormone that has been shown in many independent studies to promote tumor growth in both humans and mice.Furthermore, a low-carbohydrate, high-protein diet has the potential to both boost the ability of the immune system to kill cancer cells and prevent obesity, which leads to chronic inflammation and cancer.

SYDNEY, Aug. 25 (Xinhua) -- Scientists in Australia have discovered a way of stopping mosquitoes carrying dengue virus, raising hopes for preventing the 50 million human cases of the disease every year, local media reported on Thursday.Groundbreaking experiments in Queensland have found a common insect bacteria, wMel Wolbachia, which can dramatically reduce the presence of dengue fever in mosquitoes.The research, led by Professor Scott O'Neill, Dean at Melbourne's Monash University, was published on Thursday in the prestigious journal, Nature.Australian researchers working on the Eliminate Dengue program aim to protect the mosquitoes themselves from dengue and so stop them transmitting the virus to humans."What the experiments have shown is that this strain of Wolbachia when it is put into mosquitoes really reduces the ability of the (dengue) virus to grow in the mosquito and if it can't grow, then it can't get transmitted in people," O'Neill told reporters.O'Neill said while it was too early to say if the experiments heralded the end of dengue fever, it was a major step towards that goal.In the past decade, there have been 2400 cases of dengue fever reported during 36 outbreaks in Australia.Dengue fever has become endemic in tropical regions, where it is spread by a specific type of mosquito that becomes infected after biting humans with the disease.Despite millions of people being infected with dengue each year, there is currently no way of stopping its rapid spread either by vaccines or controlling mosquito populations.Further trials will be conducted in Cairns in north Queensland over the coming wet season and approval is currently being sought for trials in Thailand, Vietnam, Brazil and Indonesia that will directly determine the effectiveness of the method in reducing dengue disease in human populations, according to Monash University.

WASHINGTON, June 8 (Xinhua) -- Hundreds of small genetic variations are associated with autism spectrum disorders, including an area of DNA that may be a key to understanding why humans are social animals, according to a multi-site collaborative study led by researchers at Yale University.To be published Thursday in the journal Neuron, the study reinforces the theory that autism, a disorder that develops in early childhood involving impairments in social interaction, language deficits and distinctive behaviors, is not caused by one or two major genetic defects, but by many small variations, each associated with a small percentage of cases.The study looked at more than 1,000 families in which there was a single child with an autism spectrum disorder, an unaffected sibling and unaffected parents. The team compared individuals with autism to their siblings to determine what types of genetic changes distinguished the affected child from the unaffected child.One of the most intriguing of these findings points to the same small section of the genome that causes Williams syndrome -- a developmental disorder marked by high sociability and an unusual aptitude for music.In autism, there is an increase in the chromosomal material, an extra copy of this region, and in Williams syndrome, there's a loss of that same material," said lead author Matthew State. "What makes this observation particularly interesting is that Williams syndrome is known for a personality type that is highly empathetic, social, and sensitive to the emotional state of others. Individuals with autism often have difficulties in the opposite direction. This suggests that there is an important key in that region to understanding the nature of the social brain."State and his team also found about 30 other regions in the genome that are very likely contributing to autism and are focused on about six of those regions that showed the strongest evidence."We're now moving on to a second phase of the study looking at an additional 1,600 families and should be able to identify multiple new regions that are strongly implicated in autism," he said.

LOS ANGELES, July 27 (Xinhua) -- The Juno spacecraft of the National Aeronautics and Space Administration (NASA) arrived at its launch pad at the Cape Canaveral Air Force Station in Florida on Wednesday, ready for a mission to Jupiter to study the solar system, the Jet Propulsion Laboratory (JPL) said.The solar-powered, Jupiter-bound spacecraft was secured into place on top of its rocket at 10:42 a.m. EDT (7:42 a.m. PDT), said JPL in Pasadena, California.Juno will arrive at Jupiter in July 2016 and orbit its poles 33 times to learn more about the gas giant's interior, atmosphere and aurora, according to JPL."We're about to start our journey to Jupiter to unlock the secrets of the early solar system," said Scott Bolton, the mission 's principal investigator from the Southwest Research Institute in San Antonio, Texas. "After eight years of development, the spacecraft is ready for its important mission."Now that the Juno payload is atop the most powerful Atlas rocket ever made -- the United Launch Alliance Atlas V 551 -- a final flurry of checks and tests can begin and confirm that all is go for launch, according to JPL.The final series of checks begins Wednesday with an on-pad functional test. The test is designed to confirm that the spacecraft is healthy after the fueling, encapsulation and transport operations."The on-pad functional test is the first of seven tests and reviews that Juno and its flight team will undergo during the spacecraft's last 10 days on Earth," said Jan Chodas, Juno's project manager at JPL. "There are a number of remaining pre- launch activities that we still need to focus on, but the team is really excited that the final days of preparation, which we've been anticipating for years, are finally here. We are ready to go. "The launch period for Juno opens Aug. 5, 2011, and extends through Aug. 26. For an Aug. 5 liftoff, the launch window opens at 11:34 a.m. EDT (8:34 a.m. PDT) and remains open through 12:43 p.m. EDT (9:43 a.m. PDT), JPL said.Managed by JPL, the Juno mission is part of the New Frontiers Program managed at NASA's Marshall Space Flight Center in Huntsville, Alaska.

COPENHAGEN, May 29 (Xinhua) -- The number of confirmed E.coli cases has risen to 11 with at least another eight persons suspected of having the intestinal infection in Denmark, according to Denmark's National Serum Institute on Sunday.Five of the confirmed cases show symptoms of kidney failure which marks an advanced stage of the sickness, the institute said.Danish cucumbers are suspected of helping spread the E. coli bacteria believed to be causing a deadly outbreak of intestinal infection in Denmark and Germany, local media reported Sunday.The Danish cucumbers were mixed in Germany with cucumbers originating in the Netherlands, making it difficult to determine if Danish cucumbers are in fact contaminated.Denmark's Veterinary and Food Administration (DVFA) had earlier said the suspicion against Danish cucumber was "vague", but advised Danish consumers not to eat raw tomatoes, lettuces and cucumbers from Germany, and cucumber from Spain.It is now checking Danish cucumbers for traces of E. coli with results expected on Tuesday.Some Danish retailers have now removed these products from their supermarket shelves. And the Danish branch of fast-food chain McDonald's announced Saturday that it was dropping fresh cucumbers from its menu until further notice.The infection, which is food-borne, can be caused by eating raw or uncooked foods such as vegetables contaminated with the E. coli bacteria.Symptoms of infection include mild fever, bloody diarrhea and vomiting, and can last five to seven days. It can prove fatal in the very young, sick, or elderly.

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