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LOS ANGELES, May 30 (Xinhua) -- The American Academy of Pediatrics (AAP) warned on Monday that children should keep away from energy drinks which may be dangerous for minors.The AAP urged young children and teens to avoid energy drinks entirely, saying routine consumption of sports drinks should be limited or eliminated.Energy drinks include such popular brands as Red Bull, AMP and Rockstar, the AAP said in a report.These drinks, the report said, tend to be heavily caffeinated, potentially having several times the level of caffeine found in a cup of coffee.In addition, manufacturers often add sugar and herbal stimulants such as guarana and taurine to the drinks, which are popular among kids, according to the report."There's no place for energy drinks for kids," said report co-author Dr. Marcie Beth Schneider, an adolescent physician in Greenwich, Connecticut. "There's a place for sports drinks, but that place is very specific."The caffeine in energy drinks can lead to high blood pressure, high heart rate and insomnia, she said.The other ingredients can boost the power of the caffeine, she said, adding that the drinks will have a greater effect on children because they're smaller than adults."Kids don't need to have this," she said. "This is not something they should be drinking."Schneider declined to identify any energy drinks that may be better than others for kids who insist on drinking them. If kids use energy drinks because they're tired, she said, they should get more rest instead of chugging caffeine.Half of the nation's 5,448 reported caffeine overdoses in 2007 were in people under age 19, although it's not known how many of the cases were the result of energy drink consumption, according to statistics provided by the AAP.

BEIJING, June 27 (Xinhuanet) -- A "lean gene" that helps people keep slim was found closely related to diabetes and heart disease, according to a study pulished in Nature Genetics journal on Sunday.The scientists from Britain’s Medical Research Council Epidemiology Unit conducted the study.After comparing the genetic codes of more than 75,000 people, a gene called IRS1 was spotted as the decisive factor to lower the fat level in human body.But in further study, IRS1 was found putting people to a 20 percent higher risk of developing heart disease and type-2 diabetes.The study warned that people who appear slim and fit might be also at risk.But scientists suggest that the study results did not change the general message for most people."While this study pointed to genes as one factor in determining the risk of developing these conditions, it was important to remember that lifestyle factors like diet, exercise, cutting out smoking and maintaining a healthy weight also play a vital role in reducing the risk," said Ruth Loos, who led the study.

BERLIN, June 17 (Xinhua) -- German authority said on Friday first case of human spreading deadly E. coli is detected, as death toll increases to 39 worldwide.A woman working in a kitchen of a catering company was infected by E. coli from sprouts, though she didn't fall ill immediately, said Harald Kehlborn, a spokesman for the consumer protection ministry of German state Hesse.Then she spread E. coli unconsciously to another 20 people through the food she prepared, said Kehlborn.The woman later developed serious complication of hemolytic- uremic syndrome (HUS), which causes failure of kidney and nervous system.According to the data of the Robert Koch Institute, Germany's national disease control centre, the number of people who are infected has reached 3,408 in Germany and 798 people have fallen into HUS, while the infection speed is slowing down.

MANILA, June 14 (Xinhua) -- At least 200 people, mostly children, have been taken to hospitals after allegedly suffering from food poisoning at a birthday party in the northern Philippine province of Bulacan, local newspaper Manila Bulletin reported Tuesday.Most of the victims suffered from stomach and chest pains, diarrhea and vomiting, local health officer Dr. Rizalli Lucas was quoted as saying.The victims claimed the cause of their food poisoning was the spaghetti served during a birthday celebration over the weekend in a village of Calumpit town.James P. de Jesus, mayor of Calumpit, said the situation is now contained and controlled but they still have to implement health measures and to determine the cause of the alleged food poisoning.Police have started investigating the incident.

WASHINGTON, Aug. 2 (Xinhua) -- The weakness of aging is associated with leaky calcium channels inside muscle cells and a drug already in Phase II clinical trials for the treatment of heart failure might plug those leaks, according to a report published Tuesday in the online edition of Cell Metabolism.Earlier studies by the research team led by Andrew Marks of Columbia University showed the same leaks underlie the weakness and fatigue that come with heart failure and Duchenne muscular dystrophy."It's interesting, normal people essentially acquire a form of muscular dystrophy with age," Marks said. "The basis for muscle weakness is the same." Extreme exercise like that done by marathon runners also springs the same sort of leaks, he added, but in that case damaged muscles return to normal after a few days of rest. A microscopic view shows smooth muscle cells derived from human embryonic stem cells showing the nuclei (blue) and proteins of the cytoskeleton (green) in this handout photo released to Reuters by the California Institute for Regenerative Medicine, March 9, 2009The leaks occur in a calcium release channel called ryanodine receptor 1 (RyR1) that is required for muscles to contract. Under conditions of stress, those channels are chemically modified and lose a stabilizing subunit known as calstabin1.Calcium inside of muscle cells is usually kept contained. When it is allowed to leak out into the cell that calcium itself is toxic, turning on an enzyme that chews up muscle cells. Once the leak starts, it's a vicious cycle. The calcium leak raises levels of damaging reactive oxygen species, which oxidize RyR1 and worsen the leak.The researchers made their discovery by studying the skeletal muscles of young and old mice. They also showed that 6-month-old mice carrying a mutation that made their RyR1 channels leaky showed the same muscular defects and weakness characteristic of older mice.When older mice were treated with a drug known as S107, the calcium leak in their muscles slowed and the animals voluntarily showed about a 50 percent increase in the amount of time spent wheel running. Now in clinical trials for patients with heart failure, the drug is known to work by restoring the connection between costabilin and RyR1.Despite considerable effort to understand and reverse age- related muscle wasting, there are no established treatments available. The new work suggests there may be hope in approaching the problem from a different angle."Most research has focused on making more muscle mass," Marks said. "What's different here is that we are focused not on muscle mass but on muscle function. More muscle doesn't help if it is not functional."