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WASHINGTON, Sept. 26 (Xinhua) -- Older people with low blood levels of vitamin B12 markers may be more likely to have lower brain volumes and have problems with their thinking skills, according to researchers at Rush University Medical Center in Chicago.The results of the study will be published Tuesday in Neurology, the medical journal of the American Academy of Neurology.Foods that come from animals, including fish, meat, especially liver, milk, eggs and poultry are usual sources of vitamin B12.The study involved 121 older residents of the south side of Chicago over the age of 65. They had blood drawn to measure levels of vitamin B12 and B12-related markers that can indicate a B12 deficiency. The same subjects took tests measuring their memory and other cognitive skills.An average of four-and-a-half years later, MRI scans of the participants' brains were taken to measure total brain volume and look for other signs of brain damage.Having high levels of four of five markers for vitamin B12 deficiency was associated with having lower scores on the cognitive tests and smaller total brain volume."Our findings definitely deserve further examination," said Christine Tangney, associate professor at Rush University Medical Center and lead author of the study. "It's too early to say whether increasing vitamin B12 levels in older people through diet or supplements could prevent these problems, but it is an interesting question to explore. Findings from a British trial with B vitamin supplementation are also supportive of these outcomes."On the cognitive tests, the scores ranged from -2.18 to 1.42, with an average of 0.23. For each increase of one micromole per liter of homocysteine -- one of the markers of B12 deficiency -- the cognitive scores decreased by 0.03 standardized units or points.Tangney noted that the level of vitamin B12 itself in the blood was not associated with cognitive problems or loss in brain volume. "Our findings lend support for the contention that poor vitamin B12 status is a potential risk factor for brain atrophy and may contribute to cognitive impairment," said Tangney.

LOS ANGELES, June 9 (Xinhua) -- The edge of our solar system may not be smooth, but filled with a turbulent sea of magnetic bubbles approximately 100 million miles (160 million kilometers) wide, NASA's Jet Propulsion Laboratory (JPL) said on Thursday.The finding was based on observations from NASA's Voyager spacecraft, humanity's farthest deep space sentinels, said JPL in Pasadena, Los Angeles.While using a new computer model to analyze Voyager data, scientists found the sun's distant magnetic field is made up of bubbles which are created when magnetic field lines reorganize, said JPL.The new model suggests the field lines are broken up into self- contained structures disconnected from the solar magnetic field, according to JPL."The sun's magnetic field extends all the way to the edge of the solar system," said astronomer Merav Opher of Boston University. "Because the sun spins, its magnetic field becomes twisted and wrinkled, a bit like a ballerina's skirt. Far, far away from the sun, where the Voyagers are, the folds of the skirt bunch up."Like Earth, our sun has a magnetic field with a north pole and a south pole. The field lines are stretched outward by the solar wind, a stream of charged particles emanating from the star that interacts with material expelled from others in our corner of the Milky Way galaxy.Understanding the structure of the sun's magnetic field will allow scientists to explain how galactic cosmic rays enter our solar system and help define how the star interacts with the rest of the galaxy.The Voyager spacecraft, more than nine billion miles (14 billion kilometers) away from Earth, are traveling in a boundary region. In that area, the solar wind and magnetic field are affected by material expelled from other stars in our corner of the Milky Way galaxy.Launched in 1977, the Voyager twin spacecraft have been on a 33- year journey. They are en route to reach the edge of interstellar space. JPL built the spacecraft and continues to operate them.

New York, Sept. 23 (Xinhua) -- A Chinese scientist was presented a prestigious U.S. award on Friday for the discovery of artemisinin, a drug therapy for malaria that has saved millions of lives across the globe, especially in the developing world.Pharmacologist Tu Youyou, 81, became the first scientist on the Chinese mainland to win Lasker Award, known as "America's Nobels" for their knack of gaining future recognition by the Nobel committee.Tu, a scientist at the China Academy of Chinese Medical Sciences in Beijing, pioneered a new approach to malaria treatment that has benefited hundreds of millions of people and promises to benefit many times more. By applying modern techniques and rigor to a heritage provided by 5000 years of Chinese traditional practitioners, she has delivered its riches into the 21st century."Not often in the history of clinical medicine can we celebrate a discovery that has eased the pain and distress of hundreds of millions of people and saved the lives of countless numbers of people, particularly children, in over 100 countries," Lucy Shapiro, a member of the award jury and professor of Stanford University, said while describing Tu' s discovery.Shapiro said the discovery, chemical identification, and validation of artemisinin, a highly effective anti-malarial drug, is largely due to the "scientific insight, vision and dogged determination" of Professor Tu and her team. She thought Professor Tu's work has provided the world with arguably the most important pharmaceutical intervention in the last half century."The discovery of artemisinin is a gift to mankind from traditional Chinese medicine," Tu said while receiving the award. "Continuous exploration and development of traditional medicine will, without doubt, bring more medicines to the world."

WASHINGTON, June 16 (Xinhua) -- Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage. New research by a team of scientists at the University of Rochester has unveiled an important new mechanism that allows cells to recognize when they are under stress and prime the DNA repair machinery to respond to the threat of damage.Their findings will be published Friday in journal Science. Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage.The scientists, led by biologists Vera Gorbunova and Andrei Seluanov, focused on the most dangerous type of DNA damage -- double strand breaks. Unrepaired, this type of damage can lead to premature aging and cancer. They studied how oxidative stress affects efficiency of DNA repair. Oxidative stress occurs when the body is unable to neutralize the highly-reactive molecules, which are typically produced during routine cellular activities.The research team found that human cells undergoing oxidative stress synthesized more of a protein called SIRT6. By increasing SIRT6 levels, cells were able to stimulate their ability to repair double strand breaks. When the cells were treated with a drug that inactivated SIRT6, DNA repair came to a halt, thus confirming the role of SIRT6 in DNA repair. Gorbunova notes that the SIRT6 protein is structurally related to another protein, SIR2, which has been shown to extend lifespan in multiple model organisms."SIRT6 also affects DNA repair when there is no oxidative stress," explains Gorbunova. "It's just that the effect is magnified when the cells are challenged with even small amounts of oxidative stress."SIRT6 allows the cells to be economical with their resources, priming the repair enzymes only when there is damage that needs to be repaired. Thus SIRT6 may be a master regulator that coordinates stress and DNA repair activities, according to Gorbunova.

CHONGQING, May 28 (Xinhua) -- Non-infectious chronic diseases have become the major threat to human health in China as deaths from such diseases account for 85 percent of annual total deaths in the country, a report issued Saturday warned.The report said a 2008 national survey on mortality cause in China showed that the figure was up from 53 percent in 1973. Such diseases result in 3.7 million deaths annually.The Disease Prevention and Control Bureau under the Ministry of Health, and the Chinese Center for Disease Control and Prevention conducted the study, which was released at a national forum on prevention and control of chronic diseases in Chongqing.The mortality-cause survey shows that four non-infectious chronic diseases -- Cerebrovascular disease, cancer, respiratory disease and heart disease -- are the four principal causes that led to the largest number of deaths in China.Chronic diseases are the leading cause of mortality in the world, accounting for 60 percent of all deaths, reports the World Health Organization.According to the report, changes in lifestyle is one of the reasons that chronic diseases are increasing. Food with high contents of fat, protein and salt can lead to high blood pressure, high blood-fat and high blood sugar. Meanwhile, more people ride in vehicles instead of walking, meaning they exercise less.Experts at the forum called for more efforts to prevent and treat chronic diseases."It allows no delay," Kong Lingzhi, the vice director of the Disease Prevention and Control Bureau said, underlining the importance of reinforcing public education on chronic disease prevention and control."The earlier patients are aware of chronic diseases and receive proper medical treatment, the more likely they could overcome the diseases," she said.According to Kong, China has set a strategy to cope with chronic diseases in which government assumes the main responsibility, while the focus is on prevention.She said China would work to establish a prevention mechanism that pools the efforts of households, communities, professional institutions, and society at large.