吉林看阳痿早泄都有哪家医院(吉林包皮过长检查费用) (今日更新中)

WASHINGTON, Sept. 27 (Xinhua) -- At least 13 people had been killed amid 72 sickened in 18 states in listeria outbreak traced to Colorado cantaloupes, making it the most deadly U.S. outbreak of food-borne infection since 1998, the Centers for Disease Control and Prevention (CDC) said Tuesday in a statement posted on its website.Of the 13 deaths, four were in New Mexico, two were in Colorado, two were in Texas, and there was one each in Kansas, Maryland, Missouri, Nebraska and Oklahoma.Victims range in age from 35 to 96 years, with an average age of 78. All of the illnesses started on or after July 31.The figures were the latest confirmed as of Monday morning, according to the CDC. But they may well rise in the still-widening outbreak as state and local officials are investigating three additional deaths that may be connected.In 1998, 21 people died from listeria linked to tainted hot dogs, according to a CDC online database.The Food and Drug Administration on Sept. 14 warned consumers not to eat cantaloupes from Colorado's Rocky Ford region shipped by Jensen Farms. The cantaloupes with the brand name Rocky Ford were distributed from July 29 to Sept. 10 in at least 17 states.Listeria is a common bacterium that typically causes mild illness in healthy people, but can cause severe illness in older people and those with compromised immune systems. It also can cause miscarriages and stillbirths in pregnant women and severe infections in new babies.Listeria infections lead to about 1,600 serious illnesses each year and about 260 people die, according to the CDC.The CDC estimates that about 48 million people in the U.S. each year get sick from tainted food, with about 128,000 hospitalized and 3,000 deaths.

BEIJING, Sept. 9 (Xinhua) -- Lenovo Group, China's largest PC maker, said it aims to become the world's second-largest PC provider by the end of this year, China Daily reported Friday.The PC maker will take aggressive action to expand in the international PC market left by its competitors because of strategy adjustment, the English newspaper quoted Liu Chuanzhi, the board chairman of Lenovo, as saying.A few weeks ago, the world's biggest PC maker Hewlett-Packard Corp. said it will spin off its PC sector. Apple Inc's former CEO Steve Jobs resigned."The Chinese market is the starting point for Lenovo, but it won't be the only place Lenovo should put emphasis on. We will set up a more active strategy for expanding in overseas markets," Liu said.The PC maker has long been focusing on the global market. It purchased the PC division of the IBM Corp. a few years ago. In January, it announced a 175-million-U.S.-dollar joint venture with Japan's NEC Corp. In July, Lenovo completed its acquisition of Medion AG, a German multimedia and consumer electronics maker.

WASHINGTON, June 16 (Xinhua) -- Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage. New research by a team of scientists at the University of Rochester has unveiled an important new mechanism that allows cells to recognize when they are under stress and prime the DNA repair machinery to respond to the threat of damage.Their findings will be published Friday in journal Science. Cells in the human body are constantly being exposed to stress from environmental chemicals or errors in routine cellular processes. While stress can cause damage, it can also provide the stimulus for undoing the damage.The scientists, led by biologists Vera Gorbunova and Andrei Seluanov, focused on the most dangerous type of DNA damage -- double strand breaks. Unrepaired, this type of damage can lead to premature aging and cancer. They studied how oxidative stress affects efficiency of DNA repair. Oxidative stress occurs when the body is unable to neutralize the highly-reactive molecules, which are typically produced during routine cellular activities.The research team found that human cells undergoing oxidative stress synthesized more of a protein called SIRT6. By increasing SIRT6 levels, cells were able to stimulate their ability to repair double strand breaks. When the cells were treated with a drug that inactivated SIRT6, DNA repair came to a halt, thus confirming the role of SIRT6 in DNA repair. Gorbunova notes that the SIRT6 protein is structurally related to another protein, SIR2, which has been shown to extend lifespan in multiple model organisms."SIRT6 also affects DNA repair when there is no oxidative stress," explains Gorbunova. "It's just that the effect is magnified when the cells are challenged with even small amounts of oxidative stress."SIRT6 allows the cells to be economical with their resources, priming the repair enzymes only when there is damage that needs to be repaired. Thus SIRT6 may be a master regulator that coordinates stress and DNA repair activities, according to Gorbunova.

CANBERRA, Aug. 17 (Xinhua) -- Australian scientist on Wednesday said his international research team has discovered the trick on how butterfly learn to change its wing pattern to avoid being eaten by birds.The Amazonian butterfly, Heliconius numata, has learnt to carry out a single genetic switch to alter its wing pattern so it appears to be another bad-tasting butterfly that birds will avoid.Dr. Siu Fai (Ronald) Lee from the Department of Genetics and Bio21 Institute at Australia's University of Melbourne was part of the international research team, which was led by scientists at Centre National de la Recherche Scientifique (CNRS) and the University of Exeter in United Kingdom.Dr. Lee said the historical mystery had puzzled researchers for decades."Charles Darwin was puzzled by how butterflies evolved such similar patterns of warning coloration," Dr. Siu Fai (Ronald) Lee from the Department of Genetics and Bio21 Institute at the University of Melbourne told Western Australia Today."We have now solved this mystery, identifying the region of chromosome responsible for changing wing pattern."He said the research team identified a genetic switch known as a supergene, which allowed the butterfly to morph into several different forms, allowing one species to mimic another."It is amazing that by changing just one small region of the chromosomes, the butterfly is able to fool its predators by mimicking a range of different butterflies that taste bad," he said."The butterflies rearrange this supergene DNA like a small pack of cards, and the result is new wing patterns. It means that butterflies look completely different but have the same DNA."There are other butterflies doing similar tricks, but this is the most elegant one."I was just fascinated by how elegant they were."He said the discovery proves that small chromosomal changes can preserve successful gene combinations, and thus help a species to adapt.The findings of the study are published on August 14 in the international journal Nature.

concerned about weaker economic growth being a reality for most advanced economies."Further mis-steps from European and US policymakers risk converting the cracks in their economies into a much deeper global system crisis which would have worrying economic and?social consequences," he warned.But Smith, whose bank is focusing on Asia to drive profit growth, remains optimistic for the outlook in the Asia-Pacific region.He said that the fact is that Australia is incredibly well positioned because of the nation's linkage to Asia, which remains the best performing part of the global economy.His comments came after ANZ reported a 1.45 billion U.S. dollars underlying profit for the June quarter, up 1.3 percent compared to the March quarter.

来源：资阳报