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WASHINGTON, Dec. 12 (Xinhua) -- British scientists have discovered a new way to target cancer through manipulating a master switch responsible for cancer cell growth. The findings, published Monday in the U.S. journal Cancer Cell, reveal how cancer cells grow faster by producing their own blood vessels.Cancer cells gain the nutrients they need by producing proteins that make blood vessels grow, helping deliver oxygen and sugars to the tumor. These proteins are vascular growth factors like VEGF -- the target for the anti-cancer drug Avastin. Making these proteins requires the slotting together of different parts of genes, a process called splicing.Scientists at the University of West England and the University of Bristol discovered that mutations in one specific cancer gene can control how splicing is balanced, allowing a master switch in the cell to be turned on. This master switch of splicing makes cancer cells grow faster, and blood vessels to grow more quickly, as they alter how VEGFs are put together.In experimental models, the researchers found that by using new drugs that block this master switch they prevented blood vessel growth and stopped the growth of cancers."The research clearly demonstrates that it may be possible to block tumor growth by targeting and manipulating alternative splicing in patients, adding to the increasingly wide armory of potential anti-cancer therapies," the authors said.

WASHINGTON, Dec. 12 (Xinhua) -- British scientists have discovered a new way to target cancer through manipulating a master switch responsible for cancer cell growth. The findings, published Monday in the U.S. journal Cancer Cell, reveal how cancer cells grow faster by producing their own blood vessels.Cancer cells gain the nutrients they need by producing proteins that make blood vessels grow, helping deliver oxygen and sugars to the tumor. These proteins are vascular growth factors like VEGF -- the target for the anti-cancer drug Avastin. Making these proteins requires the slotting together of different parts of genes, a process called splicing.Scientists at the University of West England and the University of Bristol discovered that mutations in one specific cancer gene can control how splicing is balanced, allowing a master switch in the cell to be turned on. This master switch of splicing makes cancer cells grow faster, and blood vessels to grow more quickly, as they alter how VEGFs are put together.In experimental models, the researchers found that by using new drugs that block this master switch they prevented blood vessel growth and stopped the growth of cancers."The research clearly demonstrates that it may be possible to block tumor growth by targeting and manipulating alternative splicing in patients, adding to the increasingly wide armory of potential anti-cancer therapies," the authors said.

BEIJING, Oct. 7 (Xinhua) -- Chinese scientists are developing new methods like spraying or dropping hepatitis vaccines into people's noses to replace traditional injections, according to a leading expert on immunology.Such researches were sponsored by a key national science project, according to Wen Yumei, an academician of the Chinese Academy of Engineering, also the chairwoman of the 14th International Symposium on Viral Hepatitis and Liver Disease, which is scheduled to be held in Shanghai next July.At a press conference held here Friday for the symposium, experts believed that broad-coverage inoculations is an effective way in facilitating the country's goal in controlling Hepatitis B.China's Ministry of Health has also credited its national immunization program for having protected about 80 million people from being infected with the Hepatitis B virus (HBV) in the 19 years after 1992.Recent report showed that about 93 million Chinese are HBV carriers, accounting for more than a quarter of the world's total.

WASHINGTON, Dec. 21 (Xinhua) -- NASA's Dawn spacecraft has sent around 10,000 pictures of the giant asteroid Vesta back to Earth for the first time since its launch in 2007, the U.S. space agency announced Wednesday.The images were taken from an average altitude of 209 km, the closest the spacecraft can get.From the images, scientists could see Vesta's stippled and lumpy surface in great detail, and its many small craters, grooves, and lineaments. The fine scale of the images highlights small outcrops of bright and dark material.With these new discoveries, scientists believe that studying Vesta can help decode the early history of the solar system.Dawn will maintain its low-altitude orbit for another 10 weeks before moving to a higher orbit for another round of photo-taking.Launched in 2007, the 446 million U.S. dollar project aims at studying the chemical composition of Vesta's surface and probing its interior structure.Dawn will leave Vesta in July next year and arrive at the dwarf planet Ceres in February 2015.