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SAN FRANCISCO, April 5 (Xinhua) -- The appeal of tablet computer and electronic-book reader (e-reader) has been giving a boost to semiconductor market as sales of chips for such devices surged in 2010 and keeps growing, market research firm International Data Corp. (IDC) said on Tuesday.A new IDC report showed that worldwide revenues for media tablet and e-reader semiconductors grew by over 2,000 percent to 3. 3 billion U.S. dollars in 2010 as semiconductor suppliers enabled original equipment manufacturers to bring new products to market less than 8 months after Apple Inc. launched its iPad tablet.According to IDC's definition, media tablets are devices with color displays larger than 5-inch and smaller than 14-inch, running lightweight operating systems and able to be based on either x86 or ARM processors."The opportunity for semiconductors in media tablets and e- readers has exploded and semiconductor suppliers are scrambling to bring to market semiconductor and software platforms to enable these products," Michael Palma, a senior research analyst at IDC, said in a statement.Looking forward, IDC said it expects media tablet and e-reader semiconductor revenues to grow by 120 percent year over year in 2011, predicting that the market will be boosted by the arrival of a new version of Google Inc.'s Android operating system, dual core processors and increased bandwidth."For the next several years, we will see rapid innovation cycles for products launched into the marketplace and semiconductor suppliers will continue to satisfy evolving end user requirements over the coming years," Palma noted.

JERUSALEM, April 12 (Xinhua) -- Israeli-Canadian archaeologist Simcha Jacobovici may have stumbled upon a major archaeological find if his claim of unearthing the nails used to hold Jesus on the cross during his crucifixion are to be believed.Jacobovici maintains that small metal remains dug out of the soil in a cave in a Jerusalem neighborhood are in fact the nails that held Jesus on the cross, the Ha'aretz daily reported Tuesday.The artifacts were found in a cave in the Armon Hanatziv ridge, about 6 km south of the Old City, during the course of a construction work in 1990. The hilly area now contains apartment buildings.During the excavation of the cave, a stone vessel with the name Caiaphas, the name of the High Priest considered responsible for turning Jesus over to the Romans, and two rusty nails, which Jacobovici believes to be the relics, was unearthed.However, officials from the Israeli Antiquities Authority told Xinhua although they regard Jacobovici as a great archaeologist and documentary filmmaker, they do not believe the cave really belongs to the High Priest of the Second Temple period (between 536 BC and 70 AD)."The reason is that during that period of time there were a lot of people with the name Caiaphas," a statement from the Antiquities Authority said, "And for example, it is inconceivable that every gravestone bearing the name Moshe Dayan, which is very common in these parts, should be ascribed to the same personality who played so prominent a role in our recent history."Dayan was an Israeli army general who led his troops to victory in the 1967 war.The authority considers Jacobovici's contention as "fantasy" that should not be considered conclusive, since there is no agreed- upon evidence backing it up.Jacobovici is standing his ground, however, and contends that during the Second Temple Period the name Caiaphas was uncommon, and therefore the tomb must belong to the infamous priest.Jacobovici, a well-known documentary filmmaker, has produced films about the Israeli-Palestinian conflict and human trafficking that earned him two Emmy Awards.He also worked alongside film director James Cameron on the movie Jesus' Lost Tomb.He expects to soon release a movie about the latest finds, entitled Nails on the Cross.

BEIJING, March 15 (Xinhuanet) -- The number of Alzheimer patients is growing rapidly, so is that of unpaid caregivers, says a report released Tuesday by the Alzheimer's Association.Nearly 15 million caregivers, most of them family members but also friends, are providing billions of hours of unpaid care for Alzheimer's patients and other forms of dementia in the U.S. — 37 percent more than last year, the report says.Alzheimer's is the sixth leading cause of death in the nation, and the only one among the top 10 that have no prevention or cure, says William Thies, chief medical and scientific officer of the Alzheimer's Association.However, if people were more aware of early symptoms and were diagnosed sooner, then planning could help ease stress on patients and caregivers, according to Beth Kallmyer, senior director of constituent services of Alzheimer's Association."The toll on families is devastating," says Kallmyer, "Stress is extremely high, and one-third are experiencing depression."The time and stress of caring for an Alzheimer’s patient takes a physical toll, translating into nearly 8 billion dollars worth of extra health care costs for caregivers, the report says.

BEIJING, March 5 (Xinhua) -- China's retailers will offer refund to recent buyers of iPad 1 as price dropped with the debut of iPad 2.Apple released the upgraded version of the tablet on March 3 and announced to lower the price tags of the first generation iPad 1 by up to 27.6 percent. Buyers of iPad 1 within the last two weeks are eligible for a price difference compensation, according to Apple.Chinese customers who bought the product via Apple China online or its chain stores will also be compensated, according to a statement on Apple's website.Chinese electronics retailers such as Suning and Gome responded quickly to the price cut, promising to pay back their customers with a refund.Suning, China's biggest electronics retailer by market value, said iPad 1 buyers who bought the product from Feb 17 to March 2 can get the refund with the receipt starting Saturday.Gome, the second largest electronics retailer in China, also announced to pay back their customers who bought the product from Feb 18 to March 3 from their stores. Customers could apply for the refund with the receipt starting Sunday.

WELLINGTON, May 24 (Xinhua) -- New Zealand researchers have found a way to stop the growth of certain cancer tumors by " silencing" a group of PAX genes, members of a small family of genes that play important roles in embryonic development, but also allow cancer cells to grow and divide in adult tissue.In an article published in UK medical journal Oncogene, Otago University Professor Michael Eccles and colleagues revealed how they used the PAX8 gene to kill cancer cells.After detecting high levels of PAX8 protein in the majority of kidney, ovarian and thyroid cancers they studied, the researchers used molecular techniques to silence the PAX8 gene in several cancer cell lines."We found that these PAX8-depleted cancer cells ceased growing and dividing. The cells were essentially stopped in their tracks through the failure of multiple mechanisms and pathways crucial to their cell division cycle. They then entered into a state called senescence in which they no longer divided, and after that they ultimately died," Eccles said in a statement from the university Tuesday.The findings suggested that PAX8 could be a good target for the development of new cancer therapies, he said."Any resulting drugs would be a long way down the road, but in the meantime this research helps confirm that a focus on PAX genes may prove to be a fruitful line of attack against a number of cancers," he said.The research was supported by grants from the Health Research Council of New Zealand. It formed the main piece of work carried out by Otago doctoral graduate Caiyun (Grace) Li, now a postdoctoral fellow at Stanford University. Study co-authors also included Professor Antony Braithwaite and master's student Jen Nyman.In 2003, research led by Eccles discovered that proteins from one or more of the nine PAX genes were present in many common cancers. They found that "silencing" the gene expression of PAX2 in ovarian and bladder cancer cells, and of PAX3 in melanoma, led to the rapid death of the cells.

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