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BEIJING, June 28 (Xinhuanet) -- A truck-sized asteroid passed over the Atlantic Ocean within 7,500 miles of the Earth on Monday, according to NASA's Jet Propulsion Laboratory.The space rock, called asteroid 2011 MD, is the second space rock to zip extremely close to the Earth this year.The asteroid, with latest size estimates ranging from 16 to 66 feet (5 to 20 meters) wide, was discovered just last week on June 22, but there was never any risk of it impacting the Earth, NASA scientists said. That means asteroid 2011 MD would likely be too small to survive the fiery plunge through the Earth's atmosphere, let alone reach the surface, NASA scientists said. Asteroids this size can be expected to buzz Earth with close shaves about once every six years, they added.The space rock flew well below geosynchronous satellites, which orbit 22,236 miles (35,786 km) above the Earth, but well above the 220-mile (354-km) altitude of the International Space Station.There was little chance that the asteroid would hit a satellite because of the vastness of space and relatively small number of satellites, experts said.

BEIJING, Aug. 30 (Xinhuanet) -- The search giant Google admits it sees the new Google+ social network as an "identity service" or platform on which it can build other products, according to media reports.Google chariman Eric Schmidt said Google isn't interested in changing its policies to accommodate users such as political dissidents or others who prefer to remain anonymous: If people want to remain anonymous, then they shouldn't use Google+."Fundamentally, [Google+] depends on people using their real names if they're going to build future products that leverage that information," NPR's Andy Carvin wrote in a post on Google+ as he paraphrased Schmidt's remarks.Critics say the move is harmful to political activists, victims of harassment and numerous other groups for whom using a real name online might pose a safety risk, according to CNN reports."Regarding people who are concerned about their safety, [Schmidt] said G+ is completely optional," Carvin wrote.Meanwhile, according to Carvin, Schmidt also said "the Internet would be better if we knew you were a real person rather than a dog or a fake person. Some people are just evil and we should be able to ID them and rank them downward."

WASHINGTON, Sept. 12 (Xinhua) -- An enzyme that appears to play a role in controlling the brain's response to nicotine and alcohol in mice might be a promising target for a drug that simultaneously would treat nicotine addiction and alcohol abuse in people, U.S. researchers find.Over the course of four weeks, mice genetically engineered to lack the gene for protein kinase C (PKC) epsilon consumed less of a nicotine-containing water solution than normal mice, and were less likely to return to a chamber in which they had been given nicotine. In contrast, normal mice steadily increased their consumption of nicotine solution while the mice lacking PKC epsilon did not.The study conducted by researchers at the Ernest Gallo Clinic and Research Center, affiliated with the University of California, San Francisco, appeared Monday in the online edition of the Proceedings of the National Academy of Sciences.In normal mice, as in humans, nicotine binds to a certain class of nicotinic receptors located on dopamine neurons, which causes dopamine to be released in the brain. Dopamine creates a feeling of enjoyment, and thus prompts a sense of reward. Researchers found that mice lacking PKC epsilon are deficient in these nicotinic receptors.The finding complements earlier research in which researchers found that mice genetically engineered to lack the PKC epsilon enzyme drank less alcohol than normal mice and were disinclined to return to a chamber in which they had been given alcohol."This could mean that these mice might not get the same sense of reward from nicotine or alcohol," said Gallo senior associate director and investigator Robert Messing. "The enzyme looks like it regulates the part of the reward system that involves these nicotinic receptors."The reward system is a complex of areas in the brain that affect craving for nicotine, alcohol and other addictive substances.The next step in the research, said Messing, would be to develop compounds that inhibit PKC epsilon. The ultimate goal, he added, would be medications that could be used "to take the edge off of addiction by helping people get over some of their reward craving."